Phenotype
|
Fish
|
Conditions
|
Figures
|
notochord truncated, abnormal
|
AB + MO1-chrd
|
standard conditions
|
Fig. 7
from Dixon Fox et al., 2009
|
whole organism ventral region ved expression increased distribution, abnormal
|
AB + MO1-chrd
|
standard conditions
|
Fig 6
from Ye et al., 2019
|
whole organism wholly ventralized, abnormal
|
AB + MO1-chrd
|
standard conditions
|
Fig. 7
from Dixon Fox et al., 2009
|
pancreatic B cell increased amount, abnormal
|
AB + MO1-chrd
|
standard conditions
|
Fig. 2
from Song et al., 2007
|
notochord decreased width, abnormal
|
AB + MO1-chrd
|
standard conditions
|
Fig. 7
from Dixon Fox et al., 2009
|
whole organism ventral region szl expression increased distribution, abnormal
|
AB + MO1-chrd
|
standard conditions
|
Fig 6
from Ye et al., 2019
|
whole organism ventralized, abnormal
|
AB + MO1-chrd
|
standard conditions
|
Fig 6
from Ye et al., 2019
|
head decreased size, abnormal
|
AB + MO1-chrd
|
standard conditions
|
Fig. 7
from Dixon Fox et al., 2009
|
intermediate cell mass of mesoderm increased size, abnormal
|
TL + MO1-chrd
|
standard conditions
|
Fig. 2
from Kamachi et al., 2008
|
post-vent region increased size, abnormal
|
TL + MO1-chrd
|
standard conditions
|
Fig. 2
from Kamachi et al., 2008
|
brain anterior region decreased size, abnormal
|
TL + MO1-chrd
|
standard conditions
|
Fig. 2
from Kamachi et al., 2008
|
post-vent region increased size, abnormal
|
TL + MO1-chrd + MO2-chrd
|
standard conditions
|
Fig. 2
from Kamachi et al., 2008
|
brain anterior region decreased size, abnormal
|
TL + MO1-chrd + MO2-chrd
|
standard conditions
|
Fig. 2
from Kamachi et al., 2008
|
intermediate cell mass of mesoderm increased size, abnormal
|
TL + MO1-chrd + MO2-chrd
|
standard conditions
|
Fig. 2
from Kamachi et al., 2008
|
dorsal/ventral pattern formation disrupted, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. 7
from Branam et al., 2010
Fig. 8
from Dal-Pra et al., 2006
|
primitive heart tube inverted, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. 5
from Aamar et al., 2010
|
Kupffer's vesicle inverted, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. 6
from Aamar et al., 2010
|
determination of left/right symmetry disrupted, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. 6
from Aamar et al., 2010
|
Kupffer's vesicle bilateral, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. 6
from Aamar et al., 2010
|
post-vent region increased size, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. 5
from Xie et al., 2011
|
somite U-shaped, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. 5
from Griepenburg et al., 2015
Fig. 5
from Xie et al., 2011
|
neural plate decreased length, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. 4
from Dal-Pra et al., 2006
|
median fin fold increased amount, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. 5
from Xie et al., 2011
|
Kupffer's vesicle decreased size, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. 6
from Aamar et al., 2010
|
primitive heart tube bilateral, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. 5
from Aamar et al., 2010
|
post-vent region apoptotic, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. 5
from Robu et al., 2007
|
whole organism ventralized, abnormal
|
WT + MO1-chrd
|
control
|
Figure 4
from Cheng et al., 2019
|
determination of heart left/right asymmetry disrupted, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. 5
from Aamar et al., 2010
|
forerunner cell group decreased size, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. 7
from Aamar et al., 2010
|
heart looping disrupted, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. S4
from Aamar et al., 2010
|
blood island size, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. 5
from Griepenburg et al., 2015
|
whole organism wholly ventralized, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. 4
from Kapp et al., 2013
Fig. 8
from Zhong et al., 2011
Fig. 7
from Branam et al., 2010
Fig. 3 ,
Fig. 4
from Zhang et al., 2010
Fig. 3 ,
Fig. 7 ,
Fig. 8
from Dal-Pra et al., 2006
|
post-vent region wholly ventralized, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. 2
from Esterberg et al., 2009
|
neural plate decreased thickness, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. 4
from Dal-Pra et al., 2006
|
head decreased size, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. 8
from Dal-Pra et al., 2006
|
determination of left/right asymmetry in lateral mesoderm disrupted, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. 4
from Aamar et al., 2010
|
otic vesicle decreased size, abnormal
|
WT + MO1-chrd
|
standard conditions
|
Fig. 4
from Dal-Pra et al., 2006
|
dorsal/ventral pattern formation disrupted, abnormal
|
WT + MO1-chrd + MO4-tp53
|
standard conditions
|
Fig. 8
from Branam et al., 2010
|
post-vent region bent, abnormal
|
WT + MO1-chrd + MO4-tp53
|
standard conditions
|
Fig. 8
from Branam et al., 2010
|
post-vent region apoptotic, abnormal
|
WT + MO1-chrd + MO4-tp53
|
standard conditions
|
Fig. 5
from Robu et al., 2007
|
whole organism wholly ventralized, abnormal
|
WT + MO1-chrd + MO4-tp53
|
standard conditions
|
Fig. S6
from Moreno-Ayala et al., 2015
Fig. 8
from Branam et al., 2010
|
whole organism wholly ventralized, abnormal
|
t32231Tg + MO1-chrd
|
standard conditions
|
Fig. 3
from Chen et al., 2012
|
pancreatic B cell decreased amount, abnormal
|
hnf1bahi2169Tg/hi2169Tg + MO1-chrd
|
standard conditions
|
Fig. 2
from Song et al., 2007
|
whole organism ventralized, exacerbated
|
marcksbihb199/ihb199 + MO1-chrd
|
standard conditions
|
Fig 6
from Ye et al., 2019
|
whole organism ventral region ved expression increased distribution, abnormal
|
marcksbihb199/ihb199 + MO1-chrd
|
standard conditions
|
Fig 6
from Ye et al., 2019
|
whole organism ventral region szl expression increased distribution, abnormal
|
marcksbihb199/ihb199 + MO1-chrd
|
standard conditions
|
Fig 6
from Ye et al., 2019
|
notochord decreased width, abnormal
|
AB + MO1-chrd + MO1-gsc
|
standard conditions
|
Fig. 7
from Dixon Fox et al., 2009
|
head decreased size, abnormal
|
AB + MO1-chrd + MO1-gsc
|
standard conditions
|
Fig. 7
from Dixon Fox et al., 2009
|
notochord truncated, abnormal
|
AB + MO1-chrd + MO1-gsc
|
standard conditions
|
Fig. 7
from Dixon Fox et al., 2009
|
whole organism wholly ventralized, abnormal
|
AB + MO1-chrd + MO1-gsc
|
standard conditions
|
Fig. 7
from Dixon Fox et al., 2009
|
dorsal convergence disrupted, abnormal
|
WT + MO1-cdh2 + MO1-chrd
|
standard conditions
|
Fig. 6
from von der Hardt et al., 2007
|
lateral mesoderm mislocalised, abnormal
|
WT + MO1-cdh2 + MO1-chrd
|
standard conditions
|
Fig. 6
from von der Hardt et al., 2007
|
dorsal/ventral pattern formation disrupted, abnormal
|
WT + MO1-chrd + MO1-fstl1b
|
standard conditions
|
Fig. 8
from Dal-Pra et al., 2006
|
whole organism wholly ventralized, abnormal
|
WT + MO1-chrd + MO1-fstl1b
|
standard conditions
|
Fig. 8
from Dal-Pra et al., 2006
|
head decreased size, abnormal
|
WT + MO1-chrd + MO1-fstl1b
|
standard conditions
|
Fig. 8
from Dal-Pra et al., 2006
|
dorsal/ventral pattern formation disrupted, abnormal
|
WT + MO1-chrd + MO1-nog1
|
standard conditions
|
Fig. 8
from Dal-Pra et al., 2006
|
head anterior region agenesis, abnormal
|
WT + MO1-chrd + MO1-nog1
|
standard conditions
|
Fig. 3
from Dal-Pra et al., 2006
|
head decreased size, abnormal
|
WT + MO1-chrd + MO1-nog1
|
standard conditions
|
Fig. 8
from Dal-Pra et al., 2006
|
neural plate decreased length, abnormal
|
WT + MO1-chrd + MO1-nog1
|
standard conditions
|
Fig. 4
from Dal-Pra et al., 2006
|
head anterior region truncated, abnormal
|
WT + MO1-chrd + MO1-nog1
|
standard conditions
|
Fig. 3
from Dal-Pra et al., 2006
|
whole organism wholly ventralized, abnormal
|
WT + MO1-chrd + MO1-nog1
|
standard conditions
|
Fig. 3
from Zhang et al., 2010
Fig. 3 ,
Fig. 7 ,
Fig. 8
from Dal-Pra et al., 2006
|
neural plate decreased thickness, abnormal
|
WT + MO1-chrd + MO1-nog1
|
standard conditions
|
Fig. 4
from Dal-Pra et al., 2006
|
otic vesicle mislocalised medially, abnormal
|
WT + MO1-chrd + MO1-nog1
|
standard conditions
|
Fig. 4
from Dal-Pra et al., 2006
|
otic vesicle decreased size, abnormal
|
WT + MO1-chrd + MO1-nog1
|
standard conditions
|
Fig. 4
from Dal-Pra et al., 2006
|
whole organism wholly ventralized, abnormal
|
WT + MO1-chrd + MO1-zmiz2 + MO4-tp53
|
standard conditions
|
Fig. S6
from Moreno-Ayala et al., 2015
|
post-vent region increased thickness, abnormal
|
WT + MO1-chrd + MO2-chrdl2
|
standard conditions
|
Fig. 7
from Branam et al., 2010
|
blood circulation disrupted, abnormal
|
WT + MO1-chrd + MO2-chrdl2
|
standard conditions
|
Fig. 7
from Branam et al., 2010
|
whole organism wholly ventralized, abnormal
|
WT + MO1-chrd + MO2-chrdl2
|
standard conditions
|
Fig. 7 ,
Fig. 8
from Branam et al., 2010
|
dorsal/ventral pattern formation disrupted, abnormal
|
WT + MO1-chrd + MO2-chrdl2
|
standard conditions
|
Fig. 7
from Branam et al., 2010
|
head morphology, abnormal
|
WT + MO1-chrd + MO2-chrdl2
|
standard conditions
|
Fig. 7
from Branam et al., 2010
|
head decreased size, abnormal
|
WT + MO1-chrd + MO2-chrdl2
|
standard conditions
|
Fig. 7
from Branam et al., 2010
|
dorsal/ventral pattern formation disrupted, abnormal
|
WT + MO1-chrd + MO2-chrdl2 + MO4-tp53
|
standard conditions
|
Fig. 8
from Branam et al., 2010
|
whole organism wholly ventralized, abnormal
|
WT + MO1-chrd + MO2-chrdl2 + MO4-tp53
|
standard conditions
|
Fig. 8
from Branam et al., 2010
|
head decreased size, abnormal
|
WT + MO1-chrd + MO2-igfbp3
|
standard conditions
|
Fig. 8
from Zhong et al., 2011
|
hematopoietic system increased size, abnormal
|
WT + MO1-chrd + MO2-igfbp3
|
standard conditions
|
Fig. 8
from Zhong et al., 2011
|
post-vent region increased size, abnormal
|
WT + MO1-chrd + MO2-igfbp3
|
standard conditions
|
Fig. 8
from Zhong et al., 2011
|
whole organism wholly ventralized, abnormal
|
WT + MO1-chrd + MO2-igfbp3
|
standard conditions
|
Fig. 8
from Zhong et al., 2011
|
whole organism wholly ventralized, abnormal
|
WT + MO1-chrd + MO2-nog1
|
standard conditions
|
text only
from Dal-Pra et al., 2006
|
Kupffer's vesicle bilateral, abnormal
|
WT + MO1-chrd + MO2-sox17
|
standard conditions
|
Fig. 6
from Aamar et al., 2010
|
Kupffer's vesicle decreased size, abnormal
|
WT + MO1-chrd + MO2-sox17
|
standard conditions
|
Fig. 6
from Aamar et al., 2010
|
determination of left/right symmetry disrupted, abnormal
|
WT + MO1-chrd + MO2-sox17
|
standard conditions
|
Fig. 6
from Aamar et al., 2010
|
Kupffer's vesicle inverted, abnormal
|
WT + MO1-chrd + MO2-sox17
|
standard conditions
|
Fig. 6
from Aamar et al., 2010
|
whole organism wholly ventralized, abnormal
|
WT + MO1-chrd + MO3-bmper
|
standard conditions
|
Fig. 3
from Zhang et al., 2010
|
head decreased size, abnormal
|
WT + MO1-chrd + MO3-chrdl2
|
standard conditions
|
Fig. 7
from Branam et al., 2010
|
post-vent region increased thickness, abnormal
|
WT + MO1-chrd + MO3-chrdl2
|
standard conditions
|
Fig. 7
from Branam et al., 2010
|
head morphology, abnormal
|
WT + MO1-chrd + MO3-chrdl2
|
standard conditions
|
Fig. 7
from Branam et al., 2010
|
blood circulation disrupted, abnormal
|
WT + MO1-chrd + MO3-chrdl2
|
standard conditions
|
Fig. 7
from Branam et al., 2010
|
whole organism wholly ventralized, abnormal
|
WT + MO1-chrd + MO3-chrdl2
|
standard conditions
|
Fig. 7
from Branam et al., 2010
|
whole organism wholly ventralized, abnormal
|
WT + MO1-chrd + MO3-wnt8a + MO5-wnt8a
|
standard conditions
|
Fig. 4
from Kapp et al., 2013
|
otic vesicle formation process quality, abnormal
|
WT + MO1-chrd + MO6-fsta
|
standard conditions
|
Fig. 2
from Kwon et al., 2009
|
otic placode formation process quality, abnormal
|
WT + MO1-chrd + MO6-fsta
|
standard conditions
|
Fig. 2
from Kwon et al., 2009
|
otic vesicle decreased size, abnormal
|
WT + MO1-chrd + MO6-fsta
|
standard conditions
|
Fig. 2
from Kwon et al., 2009
|
otic vesicle formation process quality, abnormal
|
fgf8ati282a/ti282a + MO1-chrd + MO6-fsta
|
standard conditions
|
Fig. 2
from Kwon et al., 2009
|
otic placode formation process quality, abnormal
|
fgf8ati282a/ti282a + MO1-chrd + MO6-fsta
|
standard conditions
|
Fig. 2
from Kwon et al., 2009
|
whole organism cytolysis increased occurrence, abnormal
|
TL + MO1-chrd + MO1-fstl1b + MO1-nog1
|
standard conditions
|
Fig. 4
from Langdon et al., 2016
|
whole organism wholly ventralized, abnormal
|
TL + MO1-chrd + MO1-fstl1b + MO1-nog1
|
standard conditions
|
Fig. 4
from Langdon et al., 2016
|
whole organism wholly dorsalized, abnormal
|
WT + MO1-bmp1a + MO1-chrd + MO1-tll1
|
standard conditions
|
Fig. 4
from Zhang et al., 2010
|
whole organism wholly ventralized, abnormal
|
WT + MO1-chrd + MO1-fstl1b + MO1-nog1
|
standard conditions
|
Fig. 2
from Kapp et al., 2013
Fig. 8
from Dal-Pra et al., 2006
|
dorsal/ventral pattern formation disrupted, abnormal
|
WT + MO1-chrd + MO1-fstl1b + MO1-nog1
|
standard conditions
|
Fig. 8
from Dal-Pra et al., 2006
|
head decreased size, abnormal
|
WT + MO1-chrd + MO1-fstl1b + MO1-nog1
|
standard conditions
|
Fig. 8
from Dal-Pra et al., 2006
|
trigeminal placode decreased size, abnormal
|
WT + MO1-chrd + MO2-bmper + MO6-fsta
|
standard conditions
|
Fig. 3
from Kwon et al., 2009
|
otic placode formation process quality, abnormal
|
WT + MO1-chrd + MO2-bmper + MO6-fsta
|
standard conditions
|
Fig. 2
from Kwon et al., 2009
|
otic vesicle decreased size, abnormal
|
WT + MO1-chrd + MO2-bmper + MO6-fsta
|
standard conditions
|
Fig. 2
from Kwon et al., 2009
|
otic vesicle malformed, abnormal
|
WT + MO1-chrd + MO2-bmper + MO6-fsta
|
standard conditions
|
Fig. 2
from Kwon et al., 2009
|
otic vesicle formation process quality, abnormal
|
WT + MO1-chrd + MO2-bmper + MO6-fsta
|
standard conditions
|
Fig. 2
from Kwon et al., 2009
|
inner ear lacks all parts of type otolith, abnormal
|
WT + MO1-chrd + MO2-bmper + MO6-fsta
|
standard conditions
|
Fig. 2
from Kwon et al., 2009
|
post-vent region wholly ventralized, abnormal
|
WT + MO1-chrd + MO5-dlx3b + MO5-dlx4b
|
standard conditions
|
Fig. 2
from Esterberg et al., 2009
|
intermediate cell mass of mesoderm increased size, abnormal
|
WT + MO1-chrd + MO5-dlx3b + MO5-dlx4b
|
standard conditions
|
Fig. 2
from Esterberg et al., 2009
|
whole organism cytolysis increased occurrence, abnormal
|
ctsbap24bdth/+ + MO1-chrd + MO1-fstl1b + MO1-nog1 (TL)
|
standard conditions
|
Fig. 4
from Langdon et al., 2016
|
whole organism wholly dorsalized, ameliorated
|
ctsbap24bdth/+ + MO1-chrd + MO1-fstl1b + MO1-nog1 (TL)
|
standard conditions
|
Fig. 4
from Langdon et al., 2016
|
whole organism wholly ventralized, abnormal
|
ctsbap24bdth/+ + MO1-chrd + MO1-fstl1b + MO1-nog1 (TL)
|
standard conditions
|
Fig. 4
from Langdon et al., 2016
|
otic vesicle formation process quality, abnormal
|
fgf8ati282a/ti282a + MO1-chrd + MO2-bmper + MO6-fsta
|
standard conditions
|
Fig. 2
from Kwon et al., 2009
|
otic placode formation process quality, abnormal
|
fgf8ati282a/ti282a + MO1-chrd + MO2-bmper + MO6-fsta
|
standard conditions
|
Fig. 2
from Kwon et al., 2009
|
otic vesicle aplastic, abnormal
|
fgf8ati282a/ti282a + MO1-chrd + MO2-bmper + MO6-fsta
|
standard conditions
|
Fig. 2
from Kwon et al., 2009
|
whole organism wholly ventralized, abnormal
|
Df(Chr25:szl)m60/m60 + MO1-chrd (HK)
|
standard conditions
|
Fig. 1
from Dal-Pra et al., 2006
|
whole organism wholly ventralized, abnormal
|
ints6p18ahub + MO1-chrd (AB/TU)
|
standard conditions
|
Fig. 4
from Kapp et al., 2013
|
whole organism wholly dorsalized, abnormal
|
ints6p18ahub + MO1-chrd + MO3-wnt8a + MO5-wnt8a (AB/TU)
|
standard conditions
|
Fig. 4
from Kapp et al., 2013
|
whole organism wholly ventralized, abnormal
|
ints6p18ahub + MO1-chrd + MO1-fstl1b + MO1-nog1 (AB/TU)
|
standard conditions
|
Fig. 2
from Kapp et al., 2013
|
whole organism wholly ventralized, abnormal
|
brsb2; ctnnb2p1/p1 + MO1-chrd
|
standard conditions
|
Fig. 3
from Varga et al., 2007
|
post-vent region morphology, abnormal
|
brsb2; ctnnb2p1/p1 + MO1-chrd
|
standard conditions
|
Fig. 3
from Varga et al., 2007
|
whole organism wholly ventralized, abnormal
|
brsb2; ctnnb2p1/p1 + MO1-chrd + MO1-ctnnb2 + MO2-ctnnb1
|
standard conditions
|
Fig. 3
from Varga et al., 2007
|
post-vent region morphology, abnormal
|
brsb2; ctnnb2p1/p1 + MO1-chrd + MO1-ctnnb2 + MO2-ctnnb1
|
standard conditions
|
Fig. 3
from Varga et al., 2007
|