FIGURE

Figure 2

ID

ZDB-FIG-210113-10

Publication

Danesin et al., 2021 - Sulf2a controls Shh-dependent neural fate specification in the developing spinal cord

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Figure 2

V3 interneuron production is increased upon Sulf2a depletion. Side views of 48 hpf embryos. (a–f) Detection (a–e) and quantification (f) of sim1a-expressing neurons in embryos injected with ctrlMO (a, n = 34), sulf2aMO^ATG (b, n = 20) or sulf2aMO^splice (c, n = 34) and in wild-type (d, n = 10) or sulf2a−/− (e, n = 12) embryos from two independent experiments in each case. Datasets were compared with Mann Whitney’s test (two-tailed). Data are presented as mean number of cells per embryo ± s.d (**p < 0.01, ****p < 0.0001).

Expression Data

Gene:	sim1a
Fish:	WT sulf2a^ups9/ups9 WT + MO1-sulf2a WT + MO3-sulf2a
Knockdown Reagents:	MO1-sulf2a MO3-sulf2a
Anatomical Term:	spinal cord interneuron
Stage:	Long-pec

Expression Detail

Antibody Labeling

Phenotype Data

Fish:	sulf2a^ups9/ups9 WT + MO1-sulf2a WT + MO3-sulf2a
Knockdown Reagents:	MO1-sulf2a MO3-sulf2a
Observed In:	spinal cord interneuron spinal cord interneuron cell fate specification
Stage:	Long-pec

Phenotype Detail

Acknowledgments

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