FIGURE

Fig 1

ID

ZDB-FIG-190723-791

Publication

Song et al., 2019 - HDAC1-mediated repression of the retinoic acid-responsive gene ripply3 promotes second heart field development

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Fig 1

<italic>Crg</italic> mutants have a specific deficit in VCs.

(A-B) WT sibling and crg mutants at 48 hpf. Lateral views with anterior to the left. Arrow in B indicates pericardial edema. (C-D) Hearts from WT sibling and crg mutant myl7:NLS-DsRed2 embryos at 48 hpf. Frontal views. Purple alone indicates ventricle. Yellow indicates atrium. Arrows indicate arterial pole of the ventricle. (E-G) Quantification of CMs in the atria and ventricles of WT sibling and crg mutant myl7:DsRed2-NLS embryos at 36, 48, and 72 hpf. For 36 hpf, n = 14 for WT and crg mutants. For 48 hpf, n = 20 for WT and crg mutants. For 72 hpf, n = 17 for WT and crg mutants. Asterisk in all graphs indicates p<0.05 as determined by Student’s t-test. Error bars for all graphs indicate s.e.m.

Expression Data

Gene:	DsRed2
Antibody:	Ab-S46
Fish:	f2Tg hdac1^nl18/nl18; f2Tg
Anatomical Terms:	atrium atrium cardiac muscle cell cardiac ventricle cardiac ventricle cardiac muscle cell
Stage:	Long-pec

Expression Detail

Antibody Labeling

Phenotype Data

Fish:	hdac1^nl18/nl18; f2Tg
Observed In:	atrium cardiac ventricle cardiac ventricle cardiac muscle cell extension heart pericardium whole organism
Stage Range:	Prim-25 to Protruding-mouth

Phenotype Detail

Acknowledgments

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