FIGURE

Fig. 4

ID

ZDB-FIG-160218-17

Publication

Jahangiri et al., 2016 - The AP-1 transcription factor component Fosl2 potentiates the rate of myocardial differentiation from the zebrafish second heart field

Other Figures

All Figure Page

Back to All Figure Page

Fig. 4

fosl2 mutants recover from their ventricular deficit. (A,B) Confocal images of hearts from 72hpf control sibling (CTRL; A; n=12) and fosl2 (B; n=5) mutant Tg(cmlc2:DsRed2-nuc) embryos. (C) Bar graph showing the mean numbers of total, atrial and ventricular cardiomyocytes in control, fosl2^fb15-/- and fosl2^fb16-/- (n=3) embryos. Error bars represent s.d. n.s., not significant. (D,E) Confocal images of OFT regions in control (D; n=15) and fosl2 mutant (E; n=4) embryos co-stained with antibodies recognizing striated muscle (MF20, red) or OFT smooth muscle (Elastin2, ELN2, green). Scale bars: 50µm.

Expression Data

Gene:	DsRed2
Antibodies:	Ab-MF20 Ab1-elnb
Fish:	WT fosl2^fb15/fb15 f2Tg fosl2^fb15/fb15; f2Tg
Anatomical Terms:	bulbus arteriosus smooth muscle cardiac muscle cell cardiac muscle cell nucleus striated muscle cell
Stage:	Protruding-mouth

Expression Detail

Antibody Labeling

Phenotype Data

Fish:	fosl2^fb15/fb15; f2Tg
Observed In:	atrium cardiac muscle cell cardiac ventricle cardiac muscle cell
Stage:	Protruding-mouth

Phenotype Detail

Acknowledgments

This image is the copyrighted work of the attributed author or publisher, and ZFIN has permission only to display this image to its users. Additional permissions should be obtained from the applicable author or publisher of the image. Full text @ Development