FIGURE
Fig. S2
- ID
- ZDB-FIG-151214-16
- Publication
- Gokey et al., 2015 - The V-ATPase accessory protein Atp6ap1b mediates dorsal forerunner cell proliferation and left-right asymmetry in zebrafish
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Fig. S2
Developmental defects in ciliated organs ofatp6ap1ba82/a82mutants. (A) Neuromast kinocilia were labeled with acetylated-Tubulin antibodies in wild-type and atp6ap1ba82/a82 mutants at 3 and 7 dpf. (B) Measurement of kinocilia length revealed shortened kinocilia in atp6ap1ba82/a82 mutants at 3, 5 and 7 dpf. (C) Visualization of cilia in olfacotory placodes using acetylated-Tubulin antibodies at 3 dpf. Olfactory placodes were smaller in atp6ap1ba82/a82 mutants (n=7) as compared to wild-type siblings (n=7). |
Expression Data
| Antibody: | |
|---|---|
| Fish: | |
| Anatomical Terms: | |
| Stage Range: | Protruding-mouth to Days 7-13 |
Expression Detail
Antibody Labeling
Phenotype Data
| Fish: | |
|---|---|
| Observed In: | |
| Stage Range: | Protruding-mouth to Days 7-13 |
Phenotype Detail
Acknowledgments
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Reprinted from Developmental Biology, 407(1), Gokey, J.J., Dasgupta, A., Amack, J.D., The V-ATPase accessory protein Atp6ap1b mediates dorsal forerunner cell proliferation and left-right asymmetry in zebrafish, 115-30, Copyright (2015) with permission from Elsevier. Full text @ Dev. Biol.