Image
|
Figure Caption
Fig. S2 Developmental defects in ciliated organs ofatp6ap1ba82/a82mutants. (A) Neuromast kinocilia were labeled with acetylated-Tubulin antibodies in wild-type and atp6ap1ba82/a82 mutants at 3 and 7 dpf. (B) Measurement of kinocilia length revealed shortened kinocilia in atp6ap1ba82/a82 mutants at 3, 5 and 7 dpf. (C) Visualization of cilia in olfacotory placodes using acetylated-Tubulin antibodies at 3 dpf. Olfactory placodes were smaller in atp6ap1ba82/a82 mutants (n=7) as compared to wild-type siblings (n=7).
Figure Data
Acknowledgments
This image is the copyrighted work of the attributed author or publisher, and
ZFIN has permission only to display this image to its users.
Additional permissions should be obtained from the applicable author or publisher of the image.
Reprinted from Developmental Biology, 407(1), Gokey, J.J., Dasgupta, A., Amack, J.D., The V-ATPase accessory protein Atp6ap1b mediates dorsal forerunner cell proliferation and left-right asymmetry in zebrafish, 115-30, Copyright (2015) with permission from Elsevier. Full text @ Dev. Biol.