FIGURE

Fig. s1

ID

ZDB-FIG-110621-15

Publication

Langenbacher et al., 2011 - The PAF1 complex differentially regulates cardiomyocyte specification

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Fig. s1

LA961 is a mutation in ctr9. (A–D) In contrast to wild type embryos (A), LA961 mutant embryos at 2 dpf (B) exhibit pericardial edema, a lack of pigmentation, and a curved body axis. Injection of 200 pg of ctr9^WT mRNA rescues the cardiac, pigment, and body axis defects of LA961 mutants (C). Injection of 0.75 ng of ctr9MO phenocopies the LA961 mutant phenotype (D).

Expression Data

Expression Detail

Antibody Labeling

Phenotype Data

Fish:	ctr9^la961/la961 (AB) WT + MO1-ctr9
Knockdown Reagent:	MO1-ctr9
Observed In:	pericardium pigment cell pigmentation whole organism
Stage:	Long-pec

Phenotype Detail

Acknowledgments

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Reprinted from Developmental Biology, 353(1), Langenbacher, A.D., Nguyen, C.T., Cavanaugh, A.M., Huang, J., Lu, F., and Chen, J.N., The PAF1 complex differentially regulates cardiomyocyte specification, 19-28, Copyright (2011) with permission from Elsevier. Full text @ Dev. Biol.