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Fig. s1
LA961 is a mutation in ctr9. (A–D) In contrast to wild type embryos (A), LA961 mutant embryos at 2 dpf (B) exhibit pericardial edema, a lack of pigmentation, and a curved body axis. Injection of 200 pg of ctr9WT mRNA rescues the cardiac, pigment, and body axis defects of LA961 mutants (C). Injection of 0.75 ng of ctr9MO phenocopies the LA961 mutant phenotype (D).
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Reprinted from Developmental Biology, 353(1), Langenbacher, A.D., Nguyen, C.T., Cavanaugh, A.M., Huang, J., Lu, F., and Chen, J.N., The PAF1 complex differentially regulates cardiomyocyte specification, 19-28, Copyright (2011) with permission from Elsevier. Full text @ Dev. Biol.