FIGURE

Fig. 4

ID

ZDB-FIG-110128-48

Publication

Ríos et al., 2011 - Zebrafish usp39 Mutation Leads to rb1 mRNA Splicing Defect and Pituitary Lineage Expansion

Other Figures

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Fig. 4

The hypothalamic releasing hormone and dopamine signals are unaffected in usp39 mutants.

A–C and E–G: Whole-mount in situ hybridization with hypothalamic probes at 48 hpf, ventral view. (A–C) wild-type (wt) embryos. (E–G) usp39 mutant embryos. Expression of hypothalamic markers crh, gnrh2, and gnrh3 did not change. D and H: Immunocytochemistry with tyrosine hydroxylase (TH) antibody at 48 hpf, ventral view. (H) Expression of TH in usp39 mutant embryos did not change.

Expression Data

Genes:	crhb gnrh2 gnrh3 th
Antibody:	Ab4-th
Fish:	WT usp39^zf232/zf232
Anatomical Term:	hypothalamus
Stage:	Long-pec

Expression Detail

Antibody Labeling

Phenotype Data

Fish:	usp39^zf232/zf232
Observed In:	hypothalamus
Stage:	Long-pec

Phenotype Detail

Acknowledgments

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