Phenotype
|
Fish
|
Conditions
|
Figures
|
yolk increased size, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 1
from Khatri et al., 2016
|
trunk decreased thickness, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 1
from Khatri et al., 2016
|
lateral line ganglion neurod1 expression spatial pattern, ameliorated
|
AB + MO1-coasy
|
chemical treatment by environment: coenzyme A
|
Fig. 4
from Khatri et al., 2016
|
telencephalon neurog1 expression decreased distribution, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 5
from Khatri et al., 2016
|
spinal cord neuron neurog1 expression decreased distribution, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 5
from Khatri et al., 2016
|
hindbrain neurog1 expression decreased distribution, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 5
from Khatri et al., 2016
|
head edematous, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 1
from Khatri et al., 2016
|
brain morphology, ameliorated
|
AB + MO1-coasy
|
chemical treatment by injection: coenzyme A
|
Fig. 4
from Khatri et al., 2016
|
hindbrain gata2a expression decreased distribution, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 5
from Khatri et al., 2016
|
brain cell death increased process quality, abnormal
|
AB + MO1-coasy
|
control
|
Fig. S10
from Khatri et al., 2016
|
post-vent region morphology, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 4
from Khatri et al., 2016
|
brain morphology, ameliorated
|
AB + MO1-coasy
|
chemical treatment by environment: coenzyme A
|
Fig. 4
from Khatri et al., 2016
|
telencephalon neurod1 expression spatial pattern, ameliorated
|
AB + MO1-coasy
|
chemical treatment by injection: coenzyme A
|
Fig. 4
from Khatri et al., 2016
|
post-vent region morphology, abnormal
|
AB + MO1-coasy
|
chemical treatment by injection: coenzyme A
|
Fig. 4
from Khatri et al., 2016
|
post-vent region curved, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 1 ,
Fig. 2
from Khatri et al., 2016
|
lateral line ganglion neurod1 expression decreased distribution, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 4
from Khatri et al., 2016
|
somite development process quality, ameliorated
|
AB + MO1-coasy
|
chemical treatment by environment: coenzyme A
|
Fig. 2
from Khatri et al., 2016
|
lateral line ganglion neurod1 expression spatial pattern, ameliorated
|
AB + MO1-coasy
|
chemical treatment by injection: coenzyme A
|
Fig. 4
from Khatri et al., 2016
|
tail bud tbxta expression spatial pattern, ameliorated
|
AB + MO1-coasy
|
chemical treatment by environment: coenzyme A
|
Fig. 7
from Khatri et al., 2016
|
diencephalon neurog1 expression decreased distribution, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 5
from Khatri et al., 2016
|
post-vent region morphology, ameliorated
|
AB + MO1-coasy
|
chemical treatment by environment: coenzyme A
|
Fig. 2 ,
Fig. 4
from Khatri et al., 2016
|
midbrain morphology, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 1
from Khatri et al., 2016
|
midbrain hindbrain boundary neurod1 expression decreased distribution, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 4
from Khatri et al., 2016
|
tail bud tbxta expression decreased distribution, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 7
from Khatri et al., 2016
|
brain development process quality, ameliorated
|
AB + MO1-coasy
|
chemical treatment by environment: coenzyme A
|
Fig. 2
from Khatri et al., 2016
|
tegmentum neurog1 expression decreased distribution, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 5
from Khatri et al., 2016
|
somite border structure, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 1
from Khatri et al., 2016
|
anal fin decreased size, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 1
from Khatri et al., 2016
|
notochord tbxta expression spatial pattern, ameliorated
|
AB + MO1-coasy
|
chemical treatment by environment: coenzyme A
|
Fig. 7
from Khatri et al., 2016
|
midbrain hindbrain boundary neurod1 expression spatial pattern, ameliorated
|
AB + MO1-coasy
|
chemical treatment by injection: coenzyme A
|
Fig. 4
from Khatri et al., 2016
|
diencephalon gata2a expression decreased distribution, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 5
from Khatri et al., 2016
|
telencephalon neurod1 expression spatial pattern, ameliorated
|
AB + MO1-coasy
|
chemical treatment by environment: coenzyme A
|
Fig. 4
from Khatri et al., 2016
|
telencephalon neurod1 expression decreased distribution, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 4
from Khatri et al., 2016
|
post-vent region cell death increased process quality, abnormal
|
AB + MO1-coasy
|
control
|
Fig. S10
from Khatri et al., 2016
|
segmental plate myod1 expression spatial pattern, ameliorated
|
AB + MO1-coasy
|
chemical treatment by environment: coenzyme A
|
Fig. 7
from Khatri et al., 2016
|
post-vent region kinked, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 1
from Khatri et al., 2016
|
brain development process quality, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 2
from Khatri et al., 2016
|
post-vent region edematous, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 1 ,
Fig. 2
from Khatri et al., 2016
|
midbrain hindbrain boundary neurod1 expression spatial pattern, ameliorated
|
AB + MO1-coasy
|
chemical treatment by environment: coenzyme A
|
Fig. 4
from Khatri et al., 2016
|
segmental plate myod1 expression spatial pattern, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 7
from Khatri et al., 2016
|
midbrain hindbrain boundary neural rod pax2a expression decreased distribution, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 5
from Khatri et al., 2016
|
whole organism wholly dorsalized, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 1
from Khatri et al., 2016
|
somite shape, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 1
from Khatri et al., 2016
|
hindbrain morphology, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 1
from Khatri et al., 2016
|
notochord tbxta expression decreased distribution, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 7
from Khatri et al., 2016
|
somite development process quality, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 2
from Khatri et al., 2016
|
head development disrupted, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 1
from Khatri et al., 2016
|
brain morphology, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 4
from Khatri et al., 2016
|
anal fin hypoplastic, abnormal
|
AB + MO1-coasy
|
standard conditions
|
Fig. 1
from Khatri et al., 2016
|
anatomical structure EGFP expression spatial pattern, ameliorated
|
mw29Tg + MO1-coasy
|
chemical treatment by environment: coenzyme A
|
Fig. 8
from Khatri et al., 2016
|
whole organism bmpr1aa expression decreased amount, abnormal
|
mw29Tg + MO1-coasy
|
standard conditions
|
Fig. 8
from Khatri et al., 2016
|
somite EGFP expression decreased distribution, abnormal
|
mw29Tg + MO1-coasy
|
standard conditions
|
Fig. 8
from Khatri et al., 2016
|
whole organism bmpr1ab expression decreased amount, abnormal
|
mw29Tg + MO1-coasy
|
standard conditions
|
Fig. 8
from Khatri et al., 2016
|
whole organism bmpr1bb expression decreased amount, abnormal
|
mw29Tg + MO1-coasy
|
standard conditions
|
Fig. 8
from Khatri et al., 2016
|
anatomical structure EGFP expression decreased distribution, abnormal
|
mw29Tg + MO1-coasy
|
standard conditions
|
Fig. 8
from Khatri et al., 2016
|
whole organism bmpr1ab expression amount, ameliorated
|
mw29Tg + MO1-coasy
|
chemical treatment by environment: coenzyme A
|
Fig. 8
from Khatri et al., 2016
|
whole organism bmpr1bb expression amount, ameliorated
|
mw29Tg + MO1-coasy
|
chemical treatment by environment: coenzyme A
|
Fig. 8
from Khatri et al., 2016
|
eye EGFP expression spatial pattern, ameliorated
|
mw29Tg + MO1-coasy
|
chemical treatment by environment: coenzyme A
|
Fig. 8
from Khatri et al., 2016
|
whole organism Ab3-smad5 labeling decreased amount, abnormal
|
mw29Tg + MO1-coasy
|
standard conditions
|
Fig. 8
from Khatri et al., 2016
|
whole organism bmpr2a expression amount, ameliorated
|
mw29Tg + MO1-coasy
|
chemical treatment by environment: coenzyme A
|
Fig. 8
from Khatri et al., 2016
|
whole organism bmpr2a expression decreased amount, abnormal
|
mw29Tg + MO1-coasy
|
standard conditions
|
Fig. 8
from Khatri et al., 2016
|
whole organism bmpr1ba expression decreased amount, abnormal
|
mw29Tg + MO1-coasy
|
standard conditions
|
Fig. 8
from Khatri et al., 2016
|
whole organism Ab3-smad5 labeling amount, ameliorated
|
mw29Tg + MO1-coasy
|
chemical treatment by environment: coenzyme A
|
Fig. 8
from Khatri et al., 2016
|
somite EGFP expression spatial pattern, ameliorated
|
mw29Tg + MO1-coasy
|
chemical treatment by environment: coenzyme A
|
Fig. 8
from Khatri et al., 2016
|
whole organism bmpr1ba expression amount, ameliorated
|
mw29Tg + MO1-coasy
|
chemical treatment by environment: coenzyme A
|
Fig. 8
from Khatri et al., 2016
|
eye EGFP expression decreased distribution, abnormal
|
mw29Tg + MO1-coasy
|
standard conditions
|
Fig. 8
from Khatri et al., 2016
|
BMP signaling pathway process quality, ameliorated
|
sd2Tg; y1Tg + MO1-coasy
|
chemical treatment by environment: coenzyme A
|
Fig. 8
from Khatri et al., 2016
|
vasculature development process quality, ameliorated
|
sd2Tg; y1Tg + MO1-coasy
|
chemical treatment by environment: coenzyme A
|
Fig. 6
from Khatri et al., 2016
|
intersegmental vessel physical object quality, ameliorated
|
sd2Tg; y1Tg + MO1-coasy
|
chemical treatment by environment: coenzyme A
|
Fig. 6
from Khatri et al., 2016
|
nucleate erythrocyte decreased amount, abnormal
|
sd2Tg; y1Tg + MO1-coasy
|
standard conditions
|
Fig. 6
from Khatri et al., 2016
|
vasculature development process quality, abnormal
|
sd2Tg; y1Tg + MO1-coasy
|
standard conditions
|
Fig. 6
from Khatri et al., 2016
|
BMP signaling pathway decreased process quality, abnormal
|
sd2Tg; y1Tg + MO1-coasy
|
standard conditions
|
Fig. 8
from Khatri et al., 2016
|
vasculature development disrupted, abnormal
|
sd2Tg; y1Tg + MO1-coasy
|
standard conditions
|
Fig. 6
from Khatri et al., 2016
|
intersegmental vessel hypoplastic, abnormal
|
sd2Tg; y1Tg + MO1-coasy
|
standard conditions
|
Fig. 6
from Khatri et al., 2016
|