Phenotype
|
Fish
|
Conditions
|
Figures
|
whole organism decreased length, abnormal
|
AB + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 1
from Thorpe et al., 2005
|
tail bud decreased length, abnormal
|
AB + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 1
from Thorpe et al., 2005
|
tail bud morphology, abnormal
|
AB + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 4
from Thorpe et al., 2005
|
post-vent region decreased length, abnormal
|
AB + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 1
from Thorpe et al., 2005
|
post-anal tail morphogenesis disrupted, abnormal
|
AB + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 1
from Thorpe et al., 2005
|
heart looping disrupted, abnormal
|
TL + MO1-wnt8a
|
standard conditions
|
Fig. 1
from Lin et al., 2009
|
Kupffer's vesicle cilium decreased length, abnormal
|
TL + MO1-wnt8a
|
standard conditions
|
Fig. 2
from Lin et al., 2009
|
embryonic heart tube left/right pattern formation disrupted, abnormal
|
TL + MO1-wnt8a
|
standard conditions
|
Fig. 1 ,
Fig. 3
from Lin et al., 2009
|
telencephalon increased size, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 3
from Li et al., 2011
Fig. 3
from Ding et al., 2008
Fig. 3
from Gan et al., 2008
|
pronephric tubule cell increased distance pronephric tubule cell, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
chemical treatment by environment: aphidicolin, chemical treatment by environment: hydroxyurea
|
Fig. 7
from Naylor et al., 2017
|
tail bud protruding, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 3
from Ding et al., 2008
|
pronephric tubule distal region slc12a3 expression decreased distribution, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
control
|
Fig. 7
from Naylor et al., 2017
|
common myeloid progenitor spi1b expression decreased amount, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
control
|
Fig. 8
from Naylor et al., 2017
|
pronephric tubule cell increased distance pronephric tubule cell, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
control
|
Fig. 7
from Naylor et al., 2017
|
pronephric tubule distal region slc12a3 expression decreased amount, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
control
|
Fig. 7
from Naylor et al., 2017
|
pronephric tubule proximal region has number of cell, ameliorated
|
TU + MO1-wnt8a + MO2-wnt8a
|
chemical treatment by environment: 6-bromoindirubin-3'-oxime
|
Fig. 3
from Naylor et al., 2017
|
post-vent region decreased length, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 3
from Li et al., 2011
|
pronephric tubule cell population proliferation decreased occurrence, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
control
|
Fig. 6
from Naylor et al., 2017
|
whole organism decreased size, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 2
from Naylor et al., 2017
|
whole organism wholly dorsalized, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 3
from Ding et al., 2008
|
presumptive ventral mesoderm cdx4 expression decreased amount, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
control
|
Fig. 4
from He et al., 2017
|
whole organism shape, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 3
from Ding et al., 2008
|
shield increased width, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 4
from Li et al., 2011
|
erythroid lineage cell gata1a expression decreased amount, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
control
|
Fig. 8
from Naylor et al., 2017
|
pronephric tubule proximal region has fewer parts of type cell, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
control
|
Fig. 3
from Naylor et al., 2017
|
post-vent region decreased size, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 3
from Ding et al., 2008
Fig. 3
from Gan et al., 2008
|
pronephros pax2a expression decreased amount, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
control
|
Fig. 3
from Naylor et al., 2017
|
pronephric tubule Ab1-hnf1b labeling decreased amount, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
control
|
Fig. 3
from Naylor et al., 2017
|
canonical Wnt signaling pathway decreased process quality, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
control
|
Fig. 4
from He et al., 2017
|
trunk decreased length, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 3
from Li et al., 2011
|
pronephric tubule Ab1-hnf1b labeling amount, ameliorated
|
TU + MO1-wnt8a + MO2-wnt8a
|
chemical treatment by environment: 6-bromoindirubin-3'-oxime
|
Fig. 3
from Naylor et al., 2017
|
whole organism wholly anteriorized, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 3
from Gan et al., 2008
|
caudal fin morphology, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 2
from Naylor et al., 2017
|
head increased size, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 3
from Gan et al., 2008
|
trunk posterior region absent, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 2
from Naylor et al., 2017
|
pronephros pax2a expression amount, ameliorated
|
TU + MO1-wnt8a + MO2-wnt8a
|
chemical treatment by environment: 6-bromoindirubin-3'-oxime
|
Fig. 3
from Naylor et al., 2017
|
caudal fin absent, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 2
from Naylor et al., 2017
|
presumptive ventral mesoderm tbx6 expression decreased amount, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
control
|
Fig. 4
from He et al., 2017
|
pronephric tubule slc12a3 expression decreased amount, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 4
from Naylor et al., 2017
|
histone H4K20 methyltransferase activity disrupted, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 3
from Li et al., 2011
|
apoptotic process decreased occurrence, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a + MO4-tp53
|
standard conditions
|
Fig. 4
from Naylor et al., 2017
|
pronephric tubule slc12a3 expression decreased amount, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a + MO4-tp53
|
standard conditions
|
Fig. 4
from Naylor et al., 2017
|
whole organism posterior region ab1-casp3 labeling decreased amount, abnormal
|
TU + MO1-wnt8a + MO2-wnt8a + MO4-tp53
|
standard conditions
|
Fig. 4
from Naylor et al., 2017
|
whole organism znf703 expression absent, abnormal
|
WT + MO1-wnt8a
|
standard conditions
|
Fig. 4
from Dutta et al., 2015
|
neural crest cell morphology, abnormal
|
WT + MO1-wnt8a
|
standard conditions
|
Fig. 3
from Dutta et al., 2010
|
presumptive neural plate wholly anteriorized, abnormal
|
WT + MO1-wnt8a
|
standard conditions
|
Fig. 3
from Rhinn et al., 2009
|
neural keel wholly anteriorized, abnormal
|
WT + MO1-wnt8a
|
standard conditions
|
Fig. 3
from Rhinn et al., 2009
|
neural crest cell differentiation disrupted, abnormal
|
WT + MO1-wnt8a
|
standard conditions
|
Fig. 3
from Dutta et al., 2010
|
midbrain decreased size, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 7
from Lekven et al., 2001
|
presumptive mesoderm wholly dorsalized, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 6
from Lekven et al., 2001
|
forebrain increased size, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. S1
from Kagermeier-Schenk et al., 2011
|
forebrain prominent, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 7
from Lekven et al., 2001
|
whole organism wholly dorsalized, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 6
from Wylie et al., 2014
|
somite decreased amount, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 7
from Lekven et al., 2001
|
post-vent region decreased size, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 7
from Lekven et al., 2001
|
post-vent region decreased length, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. Table 1
from Shimizu et al., 2005
Fig. 7
from Lekven et al., 2001
|
whole organism dorsal region gsc expression increased distribution, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 3
from Kagermeier-Schenk et al., 2011
|
head increased size, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. Table 1
from Shimizu et al., 2005
|
telencephalon increased size, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 4
from Waxman et al., 2004
|
eye increased size, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. S1
from Kagermeier-Schenk et al., 2011
|
embryonic pattern specification disrupted, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 6
from Lekven et al., 2001
|
presumptive neural plate wholly anteriorized, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 6
from Lekven et al., 2001
|
post-vent region kinked, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. Table 1
from Shimizu et al., 2005
|
post-vent region has fewer parts of type somite, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. Table 1
from Shimizu et al., 2005
|
trunk decreased size, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 7
from Lekven et al., 2001
|
forebrain distended, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 7
from Lekven et al., 2001
|
whole organism posterior region absent, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. S1
from Kagermeier-Schenk et al., 2011
|
presumptive telencephalon foxg1a expression increased distribution, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 3
from Kagermeier-Schenk et al., 2011
|
margin tpbg1b expression decreased distribution, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. S1
from Kagermeier-Schenk et al., 2011
|
post-vent region curved ventral, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. Table 1
from Shimizu et al., 2005
|
whole organism dead, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a + MO7-wnt8a
|
standard conditions
|
Fig. 6
from Wylie et al., 2014
|
whole organism wholly dorsalized, abnormal
|
WT + MO1-wnt8a + MO2-wnt8a + MO7-wnt8a
|
standard conditions
|
Fig. 6
from Wylie et al., 2014
|
canonical Wnt signaling pathway decreased process quality, abnormal
|
cdkn2aipcas009/cas009 + MO1-wnt8a + MO2-wnt8a
|
control
|
Fig. 4
from He et al., 2017
|
presumptive ventral mesoderm tbx6 expression decreased amount, abnormal
|
cdkn2aipcas009/cas009 + MO1-wnt8a + MO2-wnt8a
|
control
|
Fig. 4
from He et al., 2017
|
presumptive ventral mesoderm cdx4 expression decreased amount, abnormal
|
cdkn2aipcas009/cas009 + MO1-wnt8a + MO2-wnt8a
|
control
|
Fig. 4
from He et al., 2017
|
canonical Wnt signaling pathway decreased process quality, abnormal
|
cdkn2aipcas009/cas009 + MO1-wnt8a + MO2-wnt8a + MO7-tp53
|
control
|
Fig. 4
from He et al., 2017
|
presumptive ventral mesoderm tbx6 expression decreased amount, abnormal
|
cdkn2aipcas009/cas009 + MO1-wnt8a + MO2-wnt8a + MO7-tp53
|
control
|
Fig. 4
from He et al., 2017
|
presumptive ventral mesoderm cdx4 expression decreased amount, abnormal
|
cdkn2aipcas009/cas009 + MO1-wnt8a + MO2-wnt8a + MO7-tp53
|
control
|
Fig. 4
from He et al., 2017
|
post-vent region aplastic, abnormal
|
tbx16b104/b104 + MO1-wnt8a + MO2-wnt8a (AB)
|
standard conditions
|
Fig. 7
from Lekven et al., 2001
|
somite aplastic, abnormal
|
tbx16b104/b104 + MO1-wnt8a + MO2-wnt8a (AB)
|
standard conditions
|
Fig. 7
from Lekven et al., 2001
|
trunk aplastic, abnormal
|
tbx16b104/b104 + MO1-wnt8a + MO2-wnt8a (AB)
|
standard conditions
|
Fig. 7
from Lekven et al., 2001
|
trunk aplastic, abnormal
|
tbxtab160/b160 + MO1-wnt8a + MO2-wnt8a (AB)
|
standard conditions
|
Fig. 7
from Lekven et al., 2001
|
somite structure, abnormal
|
tbxtab160/b160 + MO1-wnt8a + MO2-wnt8a (AB)
|
standard conditions
|
Fig. 7
from Lekven et al., 2001
|
somite decreased amount, abnormal
|
tbxtab160/b160 + MO1-wnt8a + MO2-wnt8a (AB)
|
standard conditions
|
Fig. 7
from Lekven et al., 2001
|
somite fused with somite, abnormal
|
tbxtab160/b160 + MO1-wnt8a + MO2-wnt8a (AB)
|
standard conditions
|
Fig. 7
from Lekven et al., 2001
|
post-vent region aplastic, abnormal
|
tbxtab160/b160 + MO1-wnt8a + MO2-wnt8a (AB)
|
standard conditions
|
Fig. 7
from Lekven et al., 2001
|
tail bud decreased length, abnormal
|
AB + MO1-wnt3a + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 1
from Thorpe et al., 2005
|
somite decreased amount, abnormal
|
AB + MO1-wnt3a + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 4
from Thorpe et al., 2005
|
post-anal tail morphogenesis disrupted, abnormal
|
AB + MO1-wnt3a + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 1
from Thorpe et al., 2005
|
notochord truncated, abnormal
|
AB + MO1-wnt3a + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 4
from Thorpe et al., 2005
|
central canal dilated, abnormal
|
AB + MO1-wnt3a + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 4
from Thorpe et al., 2005
|
tail bud bent, abnormal
|
AB + MO1-wnt3a + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 4
from Thorpe et al., 2005
|
post-vent region decreased length, abnormal
|
AB + MO1-wnt3a + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 1
from Thorpe et al., 2005
|
whole organism decreased length, abnormal
|
AB + MO1-wnt3a + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 1
from Thorpe et al., 2005
|
tail bud morphology, abnormal
|
AB + MO1-wnt3a + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 4
from Thorpe et al., 2005
|
Kupffer's vesicle cilium decreased length, abnormal
|
TL + MO1-wnt3a + MO1-wnt8a
|
standard conditions
|
Fig. 2
from Lin et al., 2009
|
embryonic heart tube left/right pattern formation disrupted, abnormal
|
TL + MO1-wnt3a + MO1-wnt8a
|
standard conditions
|
Fig. 1
from Lin et al., 2009
|
heart looping disrupted, abnormal
|
TL + MO1-wnt3a + MO1-wnt8a
|
standard conditions
|
Fig. 1
from Lin et al., 2009
|
canonical Wnt signaling pathway decreased process quality, abnormal
|
TU + MO1-cdkn2aip + MO1-wnt8a + MO2-wnt8a
|
control
|
Fig. 4
from He et al., 2017
|
presumptive ventral mesoderm tbx6 expression decreased amount, abnormal
|
TU + MO1-cdkn2aip + MO1-wnt8a + MO2-wnt8a
|
control
|
Fig. 4
from He et al., 2017
|
presumptive ventral mesoderm cdx4 expression decreased amount, abnormal
|
TU + MO1-cdkn2aip + MO1-wnt8a + MO2-wnt8a
|
control
|
Fig. 4
from He et al., 2017
|
canonical Wnt signaling pathway decreased process quality, abnormal
|
TU + MO1-cdkn2aip + MO1-wnt8a + MO2-wnt8a + MO7-tp53
|
control
|
Fig. 4
from He et al., 2017
|
presumptive ventral mesoderm tbx6 expression decreased amount, abnormal
|
TU + MO1-cdkn2aip + MO1-wnt8a + MO2-wnt8a + MO7-tp53
|
control
|
Fig. 4
from He et al., 2017
|
presumptive ventral mesoderm cdx4 expression decreased amount, abnormal
|
TU + MO1-cdkn2aip + MO1-wnt8a + MO2-wnt8a + MO7-tp53
|
control
|
Fig. 4
from He et al., 2017
|
post-vent region decreased length, abnormal
|
TU + MO1-kmt5aa + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 3
from Li et al., 2011
|
telencephalon increased size, abnormal
|
TU + MO1-kmt5aa + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 3
from Li et al., 2011
|
trunk decreased length, abnormal
|
TU + MO1-kmt5aa + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 3
from Li et al., 2011
|
telencephalon increased size, abnormal
|
WT + MO1-dact1 + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 4
from Waxman et al., 2004
|
head decreased size, abnormal
|
WT + MO1-dact1 + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 4
from Waxman et al., 2004
|
post-vent region decreased size, abnormal
|
WT + MO1-dact1 + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 4
from Waxman et al., 2004
|
whole organism wholly anteriorized, abnormal
|
WT + MO1-dact1 + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. 4
from Waxman et al., 2004
|
whole organism frzb expression amount, ameliorated
|
WT + MO1-nanog + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig 3
from He et al., 2020
|
whole organism emx1 expression amount, ameliorated
|
WT + MO1-nanog + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig 3
from He et al., 2020
|
whole organism dkk1b expression amount, ameliorated
|
WT + MO1-nanog + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig 3
from He et al., 2020
|
whole organism six3b expression increased amount, abnormal
|
WT + MO1-nanog + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig 3
from He et al., 2020
|
whole organism sp5l expression position, ameliorated
|
WT + MO1-nanog + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig 3
from He et al., 2020
|
post-vent region has fewer parts of type somite, abnormal
|
WT + MO1-wnt3a + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. Table 1
from Shimizu et al., 2005
|
post-vent region truncated, abnormal
|
WT + MO1-wnt3a + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. Table 1
from Shimizu et al., 2005
|
head increased size, abnormal
|
WT + MO1-wnt3a + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. Table 1
from Shimizu et al., 2005
|
post-vent region decreased length, abnormal
|
WT + MO1-wnt3a + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. Table 1
from Shimizu et al., 2005
|
post-vent region kinked, abnormal
|
WT + MO1-wnt3a + MO1-wnt8a + MO2-wnt8a
|
standard conditions
|
Fig. Table 1
from Shimizu et al., 2005
|
post-vent region decreased length, abnormal
|
WT + MO1-wnt8a + MO2-wnt3a + MO2-wnt8a
|
standard conditions
|
Fig. Table 1
from Shimizu et al., 2005
|
head increased size, abnormal
|
WT + MO1-wnt8a + MO2-wnt3a + MO2-wnt8a
|
standard conditions
|
Fig. Table 1
from Shimizu et al., 2005
|
post-vent region has fewer parts of type somite, abnormal
|
WT + MO1-wnt8a + MO2-wnt3a + MO2-wnt8a
|
standard conditions
|
Fig. Table 1
from Shimizu et al., 2005
|
post-vent region kinked, abnormal
|
WT + MO1-wnt8a + MO2-wnt3a + MO2-wnt8a
|
standard conditions
|
Fig. Table 1
from Shimizu et al., 2005
|
post-vent region truncated, abnormal
|
WT + MO1-wnt8a + MO2-wnt3a + MO2-wnt8a
|
standard conditions
|
Fig. Table 1
from Shimizu et al., 2005
|
head truncated, abnormal
|
WT + MO1-foxa3 + MO1-gsc + MO1-wnt8a
|
standard conditions
|
Fig. 6
from Seiliez et al., 2006
|
eye fused with eye, abnormal
|
WT + MO1-foxa3 + MO1-gsc + MO1-wnt8a
|
standard conditions
|
Fig. 6
from Seiliez et al., 2006
|