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Fig. 3
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Fig. 3 Dnmt3 morphants are defective in retinal differentiation. Whole mount in situ for pax6.2, crx, and neurod in dnmt3 and control morphants at 80 hpf. Whereas all the retinal cells expressed pax6.2, a marker of retinal progenitors, markers of specific progenitors (crx and neurod) were absent in dnmt3 morphants. These defects can be complemented by co-injection of the wild-type Dnmt3 (Dnmt3WT) but not with the catalytically inactive Dnmt3 derivative (Dnmt3C1240S).
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Full text @ J. Biol. Chem.