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ZDB-FIG-110624-50

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Putiri et al., 2011 - The zebrafish maternal-effect gene mission impossible encodes the DEAH-box helicase Dhx16 and is essential for the expression of downstream endodermal genes

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Fig. s4

Spatial distribution of dorsal- and ventral-specific genes appears normal in maternally mutant mis embryos. Wild-type (A,C,E,G,I) and mis mutant (B,D,F,H,J) embryos exhibit normally restricted dorsal expression of nieuwkoid (A,B), goosecoid (C,D), foxb1.2 (E,F), and chordin (G,H), as well as normally restricted ventral expression of even skipped (I,J). (A–D) are embryos at 5 h.p.f., (E–F) at 7.5 h.p.f., and (G-J) at 8 h.p.f. All panels are animal pole views with dorsal to the right. Even though goosecoid expression is normally restricted, its levels of expression often appear reduced compared to wild-type. We have not further explored this effect, although it may be related to a requirement of mis function in the activation of additional nodal target genes such as goosecoid (Chen and Schier, 2001).

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Acknowledgments

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Reprinted from Developmental Biology, 353(2), Putiri, E., and Pelegri, F., The zebrafish maternal-effect gene mission impossible encodes the DEAH-box helicase Dhx16 and is essential for the expression of downstream endodermal genes, 275-289, Copyright (2011) with permission from Elsevier. Full text @ Dev. Biol.