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Phenotype Figure Summary
Marker:
pes
Conditions:
Standard or Control
Publication
Data
Fish
Anatomy
Provost
et al.
, 2012
Fig. 6
pes
hi2Tg/hi2Tg
; jh1Tg; jh2Tg
, 
pes
hi2Tg/hi2Tg
exocrine pancreas decreased size, abnormal
exocrine pancreas development disrupted, abnormal
eye decreased size, abnormal
head decreased size, abnormal
head opacity, abnormal
post-vent region curved, abnormal
Simmons
et al.
, 2012
Fig. 1
pes
vu166/vu166
; vu12Tg
, 
pes
vu166/+
; pes
hi2Tg/+
; vu12Tg
eye decreased size, abnormal
forebrain morphology, abnormal
mandibular arch skeleton decreased size, abnormal
otolith morphology, abnormal
pectoral fin morphology, abnormal
spinal cord dorsal region has fewer parts of type oligodendrocyte, abnormal
whole organism decreased length, abnormal
Fig. 2
pes
vu166/vu166
; vu12Tg
oligodendrocyte decreased amount, abnormal
oligodendrocyte distributed, abnormal
spinal cord dorsal region has fewer parts of type oligodendrocyte, abnormal
spinal cord grey matter has extra parts of type oligodendrocyte, abnormal
spinal cord ventral region has extra parts of type oligodendrocyte, abnormal
spinal cord white matter has fewer parts of type oligodendrocyte, abnormal
Fig. 3
pes
vu166/vu166
(AB)
central nervous system has fewer parts of type oligodendrocyte myelin sheath, abnormal
oligodendrocyte decreased amount, abnormal
oligodendrocyte differentiation disrupted, abnormal
oligodendrocyte distributed, abnormal
Fig. 4
pes
vu166/vu166
; vu12Tg
apoptotic process increased occurrence, abnormal
cell cycle process disrupted, abnormal
cell population proliferation increased occurrence, abnormal
spinal cord apoptotic, abnormal
Fig. 5
pes
vu166/vu166
(AB)
mitotic S phase arrested, abnormal
neurectodermal cell increased amount, abnormal
oligodendrocyte decreased amount, abnormal
oligodendrocyte differentiation disrupted, abnormal
Fig. 6
pes
vu166/vu166
(AB)
, 
pes
vu166/vu166
; vu16Tg
oligodendrocyte axon misrouted, abnormal
oligodendrocyte axon split, abnormal
oligodendrocyte axonal growth cone collapsed, abnormal
oligodendrocyte plasma membrane disorganized, abnormal
radial glial cell cell projection disorganized, abnormal
spinal cord has fewer parts of type radial glial cell cell projection, abnormal
Barresi
et al.
, 2010
Fig. 3
pes
hi2Tg/hi2Tg
(AB/TU)
anterior commissure astrocyte mislocalised, abnormal
anterior commissure morphology, abnormal
cranial nerve II decreased size, abnormal
cranial nerve II decreased thickness, abnormal
cranial nerve II defasciculated, abnormal
forebrain commissural neuron axon guidance disrupted, abnormal
postoptic commissure astrocyte mislocalised, abnormal
postoptic commissure morphology, abnormal
Phenotype Annotation (1994-2006)
Fig. for (hi2)
pes
hi2Tg
(AB)
anatomical system quality, abnormal
brain quality, abnormal
eye decreased size, abnormal
fin decreased size, abnormal
gut quality, abnormal
liver quality, abnormal
Amsterdam
et al.
, 2004
text only
pes
hi2Tg/hi2Tg
eye decreased size, abnormal
gut hypoplastic, abnormal
head decreased size, abnormal
liver decreased size, abnormal
pericardium edematous, abnormal
yolk circular, abnormal
yolk grey, abnormal
Allende
et al.
, 1996
Fig. 10
pes
hi2Tg/hi2Tg
gut decreased size, abnormal
liver decreased size, abnormal
pancreas aplastic, abnormal
trunk musculature degenerate, abnormal
yolk increased size, abnormal
Fig. 2
pes
hi2Tg/hi2Tg
neurocranium decreased length, abnormal
neurocranium increased thickness, abnormal
optic tectum decreased size, abnormal
Fig. 9
pes
hi2Tg/hi2Tg
ceratohyal cartilage oriented, abnormal
hyomandibula decreased size, abnormal
Meckel's cartilage fused with Meckel's cartilage, abnormal
whole organism lacks all parts of type pharyngeal arch 3-7 skeleton, abnormal
Gaiano
et al.
, 1996
Fig. 1
pes
hi2Tg/hi2Tg
whole organism edematous, abnormal
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