Publication |
Data |
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Fish |
Anatomy
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Virtanen et al., 2023
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Fig. 3
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cald1bsa24667/sa24667
, 
cald1bsa24667/+
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Zheng et al., 2009
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Fig. 1
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WT + MO1-cald1b
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Fig. 2
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WT + MO1-cald1b
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- blood circulation disrupted, abnormal
- blood vessel endothelial cell migration disrupted, abnormal
- blood vessel morphogenesis disrupted, abnormal
- caudal vein plexus decreased size, abnormal
- dorsal aorta morphology, abnormal
- dorsal longitudinal anastomotic vessel aplastic, abnormal
- intersegmental vessel decreased amount, abnormal
- intersegmental vessel morphology, abnormal
- morphogenesis of an epithelial sheet disrupted, abnormal
- nucleate erythrocyte aggregated, abnormal
- nucleate erythrocyte fluid flow rate, abnormal
- posterior cardinal vein morphology, abnormal
- subintestinal vein morphology, abnormal
- trunk vasculature morphology, abnormal
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Fig. 4
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WT + MO1-cald1b
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Fig. 5
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WT + MO1-cald1b
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text only
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WT + MO1-cald1b
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Zheng et al., 2009
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Fig. 1
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WT + MO1-cald1b
, 
WT + MO2-cald1b
|
- atrioventricular valve aplastic, abnormal
- atrium elongated, abnormal
- atrium increased size, abnormal
- cardiac muscle cell decreased amount, abnormal
- cardiac muscle cell decreased size, abnormal
- cardiac muscle cell proliferation disrupted, abnormal
- cardiac ventricle elongated, abnormal
- heart looping disrupted, abnormal
- heart morphogenesis disrupted, abnormal
- heart morphology, abnormal
- pericardium edematous, abnormal
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text only
|
|
WT + MO1-cald1b
, 
WT + MO2-cald1b
|
|
Zheng et al., 2009
|
Fig. 1
|
|
WT + MO1-cald1b
, 
WT + MO2-cald1b
|
|
Zheng et al., 2009
|
Fig. 1
|
|
WT + MO1-cald1b
, 
WT + MO2-cald1b
|
|