Publication	Data		Fish	Anatomy
Lencer et al., 2023	Fig. 3		bocco24/co24; st1002Tg	neural tube Wnt signaling pathway decreased occurrence, abnormal slow muscle cell Wnt signaling pathway decreased occurrence, abnormal
Kearns et al., 2021	Fig. 1		bocty54z/+; bocco24/+; vu12Tg/vu12Tg ,   bocco24/co24; vu12Tg/vu12Tg	oligodendrocyte decreased amount, abnormal oligodendrocyte EGFP expression decreased amount, abnormal whole organism curved, abnormal
	Fig. 2		bocco24/co24; vu12Tg/vu12Tg	motor neuron ab2-isl labeling decreased amount, abnormal motor neuron decreased amount, abnormal oligodendrocyte mbpa expression decreased amount, abnormal oligodendrocyte myrf expression decreased amount, abnormal oligodendrocyte ab1-sox10 labeling decreased amount, abnormal oligodendrocyte decreased amount, abnormal oligodendrocyte EGFP expression decreased amount, abnormal
	Fig. 3		bocco24/co24	spinal cord dorsal region olig2 expression decreased amount, abnormal spinal cord ventral region nkx2.2a expression decreased amount, abnormal
	Fig. 4		bocco24/co24; vu12Tg/vu12Tg	oligodendrocyte EGFP expression decreased amount, abnormal spinal cord ab5-sox2 labeling decreased amount, abnormal spinal cord oligodendrocyte cell fate commitment decreased process quality, abnormal
	Fig. 5		bocco24/co24; st1001Tg/st1001Tg; vu12Tg/vu12Tg	oligodendrocyte EGFP expression decreased amount, abnormal oligodendrocyte mCherry expression decreased amount, abnormal
	Fig. 6		bocco24/co24	spinal cord olig2 expression decreased amount, abnormal spinal cord adaxial-abaxial axis boc expression spatial pattern, abnormal spinal cord adaxial-abaxial axis ptch2 expression spatial pattern, abnormal spinal cord ventral region boc expression decreased amount, abnormal
	Fig. 7		bocco24/co24	regulation of smoothened signaling pathway decreased process quality, abnormal spinal cord oligodendrocyte cell fate commitment decreased process quality, abnormal spinal cord ventral region olig2 expression decreased amount, abnormal spinal cord ventral region ptch2 expression decreased amount, abnormal
Bergeron et al., 2011	Fig. 1		bocty54z/ty54z	axon extension involved in axon guidance process quality, abnormal cranial nerve II decreased length, abnormal cranial nerve II misrouted, abnormal diencephalon glial cell mislocalised, abnormal forebrain physical object quality, abnormal forebrain regionalization process quality, abnormal postoptic commissure absent, abnormal retinal ganglion cell axon guidance process quality, abnormal
	Fig. 2		bocty54z/ty54z	regulation of smoothened signaling pathway process quality, abnormal
	Fig. 4		bocty54z/ty54z ,   WT + MO1-boc	axon extension involved in axon guidance process quality, abnormal cranial nerve II misrouted, abnormal midbrain ventral region physical object quality, abnormal post-vent region curved ventral, abnormal retinal ganglion cell axon guidance process quality, abnormal trunk curved ventral, abnormal
	Fig. 5		bocty54z/ty54z ,   bocty54z/ty54z; vu17Tg	regulation of smoothened signaling pathway process quality, abnormal
	Fig. 6		vu17Tg + MO1-boc	forebrain development process quality, abnormal forebrain physical object quality, abnormal lateral floor plate physical object quality, abnormal regulation of smoothened signaling pathway process quality, abnormal spinal cord dorsal/ventral patterning process quality, abnormal
	Fig. S2		bocty54z/ty54z	adenohypophysis development process quality, abnormal adenohypophysis physical object quality, abnormal embryonic viscerocranium morphogenesis process quality, abnormal neurocranial trabecula malformed, abnormal skeletal muscle fiber development process quality, abnormal slow muscle cell decreased amount, abnormal ventral mandibular arch increased width, abnormal ventral mandibular arch malformed, abnormal
Phenotype Annotation (1994-2006)	Fig. for (ty54z)		bocty54z/ty54z	retinal ganglion cell axon guidance quality, abnormal whole organism lethal (sensu genetics), abnormal
Connor et al., 2005	Fig. 5		WT + MO1-boc ,   WT + MO2-boc	axon guidance disrupted, abnormal caudal commissure axon mislocalised, abnormal caudal commissure defasciculated, abnormal dorsal/ventral axon guidance disrupted, abnormal dorsoventral diencephalic tract morphology, abnormal forebrain development disrupted, abnormal
Karlstrom et al., 1996	Fig. 4		bocty54z/ty54z	postoptic commissure aplastic, abnormal retinal ganglion cell axon guidance process quality, abnormal retinal ganglion cell axon misrouted, abnormal tract of the postoptic commissure axon mislocalised laterally, abnormal