Publication	Data		Fish	Anatomy
Hatzold et al., 2023	Fig 1		clint1ahi1520Tg/hi1520Tg	epidermis potentially cancerous lesions increased amount, abnormal
Phatak et al., 2018	Fig. 1		zf106Tg + MO1-clint1a ,   WT + MO1-clint1a	head epidermal cell morphology, abnormal peridermal cell perinucleolar compartment EGFP expression increased amount, abnormal trunk epidermal cell morphology, abnormal
	Fig. 2		zf106Tg + MO1-clint1a	peridermal cell endocytosis increased occurrence, abnormal peridermal cell lysosome increased amount, abnormal peridermal cell perinucleolar compartment ab1-cav1 labeling increased amount, abnormal peridermal cell perinucleolar compartment EGFP expression increased amount, abnormal
	Fig. 3		zf106Tg + MO1-clint1a	epidermis cell population proliferation increased occurrence, abnormal peridermal cell cell population proliferation increased occurrence, abnormal peridermal cell decreased size, abnormal
	Fig. 4		zf106Tg + MO1-clint1a	peridermal cell endocytosis increased occurrence, abnormal
	Fig. 5		zf106Tg + MO1-clint1a	peridermal cell decreased size, abnormal
	Fig. 6		zf106Tg + MO1-clint1a	epidermis cell population proliferation increased occurrence, abnormal peridermal cell cell population proliferation increased occurrence, abnormal
	Fig. 7		zf106Tg + MO1-clint1a	head epidermal cell morphology, abnormal trunk epidermal cell morphology, abnormal
Walters et al., 2010	Fig. 6		WT + MO2-clint1a	caudal fin epidermis hyperplastic, abnormal neutrophil chemotaxis disrupted, abnormal
Dodd et al., 2009	Fig. 1		clint1ahi1520Tg/hi1520Tg (AB)	dorsal fin fold neutrophil mislocalised, abnormal epidermis malformed, abnormal inflammatory response increased occurrence, abnormal ventral fin fold neutrophil mislocalised, abnormal
	Fig. 2		clint1ahi1520Tg/hi1520Tg (AB)	cell death increased rate, abnormal epithelial cell proliferation increased rate, abnormal inflammatory response increased occurrence, abnormal leukocyte migration involved in inflammatory response increased occurrence, abnormal ventral fin fold neutrophil mislocalised, abnormal
	Fig. 3		clint1ahi1520Tg/hi1520Tg (AB)	epidermis neutrophil mislocalised, abnormal ventral fin fold neutrophil mislocalised, abnormal
	Fig. 5		AB + MO2-clint1a	epidermis malformed, abnormal epithelial cell proliferation increased rate, abnormal ventral fin fold leukocyte mislocalised, abnormal
	Fig. 6		clint1ahi1520Tg/hi1520Tg (AB)	epidermis malformed, abnormal ventral fin fold malformed, abnormal
	Fig. 7		clint1ahi1520Tg/hi1520Tg (AB)	apoptotic chromosome condensation increased occurrence, abnormal epidermis hemidesmosome decreased amount, abnormal epidermis malformed, abnormal
Kishi et al., 2008	text only		clint1ahi1520Tg/hi1520Tg	cellular senescence increased magnitude, abnormal eye decreased size, abnormal gut decreased size, abnormal head decreased size, abnormal integument texture, abnormal liver decreased size, abnormal pericardium edematous, abnormal whole organism decreased length, abnormal whole organism decreased size, abnormal yolk present, abnormal
Phenotype Annotation (1994-2006)	Fig. for (hi1520)		clint1ahi1520Tg	eye decreased size, abnormal gut quality, abnormal heart quality, abnormal liver quality, abnormal surface structure quality, abnormal whole organism decreased length, abnormal
	Fig. for (hi2558)		clint1ahi2558Tg	eye decreased size, abnormal gut quality, abnormal heart quality, abnormal liver quality, abnormal surface structure quality, abnormal whole organism decreased length, abnormal
Amsterdam et al., 2004	text only		clint1ahi1520Tg/hi1520Tg	eye decreased size, abnormal gut hypoplastic, abnormal head decreased size, abnormal integument rugose, abnormal liver hypoplastic, abnormal pericardium edematous, abnormal trunk decreased length, abnormal trunk decreased size, abnormal yolk non-functional, abnormal