ZFIN Image: Gessler et al., 2022, Figure 2

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Figure Caption

Figure 2

The involvement of adenosine deaminase family members in zebrafish pronephros repair. (A) In situ hybridization revealed upregulation of zebrafish ada. Depletion of zebrafish ada by a splice—(SBM) blocking morpholino oligonucleotides (MO) delayed the repair process. (B) In situ hybridization revealed upregulation of zebrafish ada2a. Depletion of zebrafish ada2a by a translation- (TBM) blocking morpholino oligonucleotides (MO) had no effect on the repair process. (C) In situ hybridization revealed upregulation of zebrafish ada2b. Depletion of zebrafish ada2b by a TBM delayed the repair process. (D) In situ hybridization revealed upregulation of zebrafish adal. Depletion of zebrafish adal by a TBM had no significant influence on the repair process (mean ± SEM; 2-way ANOVA).

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Acknowledgments

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