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Fig. 1
RA signaling directly regulates nr2f1a expression.
(A-C) In situ hybridization (ISH) for nr2f1a in control (ctrl), DEAB-treated, and RA-treated embryos at the 8s stage. (D-G) ISH for nr2f1a in the LPM of ctrl, RA, CHX, and RA+CHX treated embryos. (H) RT-qPCR of nr2f1a expression after RA and CHX treatments at the 3s stage. Asterisks in all graphs indicate statistically significant difference from controls with p<0.05.
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