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Fig. 6 Formation of pancreatic mesenchyme is independent of isl gene function. Neither the loss of all three isl-genes nor absence of vascularization in cloche mutants results in a loss of fgf10, fgf24 or meis3. Expression patterns of meis3 (A–D), fgf10 (E–H) and fgf24 (I–L) in pancreatic mesenchyme of control embryos (B, F, J), isl1/2a/2b depleted embryos (C, G, K) and cloche mutants (D, H, L). All images are ventral with the exception of A, E, I which are dorsal views. Anterior is to the left. Insets in A, E, I mark the position of the higher resolution images shown on the right side.
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Reprinted from Developmental Biology, 378(1), Wilfinger, A., Arkhipova, V., and Meyer, D., Cell type and tissue specific function of islet genes in zebrafish pancreas development, 25-37, Copyright (2013) with permission from Elsevier. Full text @ Dev. Biol.