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Fig. 7
Injection of suboptimal doses of ccm3a/b ATG morpholino into ccm2/vtn mutants results in very severe cranial vascular defects. Gross dilations of the AceV, PrA and MsV vessels were observed upon injection of 0.25 ng ccm3a/b ATG morpholino into ccm2/vtn embryos (A–H, yellow arrows).
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Reprinted from Developmental Biology, 362(2), Yoruk, B., Gillers, B.S., Chi, N.C., and Scott, I.C., Ccm3 functions in a manner distinct from Ccm1 and Ccm2 in a zebrafish model of CCM vascular disease, 121-131, Copyright (2012) with permission from Elsevier. Full text @ Dev. Biol.