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Fig. 3 Bilateral populations of cardiomyocytes are normal in nkx-deficient embryos. In situ hybridization depicts expression of cmlc2 (A,B) and vmhc (C,D) in wild-type (A,C) and nkx-deficient (B,D) embryos. Embryos viewed dorsally, anterior to the top, at the 18-somite stage. Aside from a mild delay in cardiomyocyte migration toward the midline, nkx-deficient embryos appear to have the same patterns of cmlc2 and vmhc expression as are observed in wild-type embryos.
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Reprinted from Developmental Biology, 322(2), Targoff, K.L., Schell, T., and Yelon, D., Nkx genes regulate heart tube extension and exert differential effects on ventricular and atrial cell number, 314-321, Copyright (2008) with permission from Elsevier. Full text @ Dev. Biol.