FIGURE

Fig. 3

ID

ZDB-FIG-110405-21

Publication

Cui et al., 2011 - Disruption of planar cell polarity activity leads to developmental biliary defects

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Fig. 3

Abnormal liver localization in pk1a morphants. Whole-mount in situ hybridization of ceruloplasmin (cp), a liver marker, in 3 dpf pk1a morphants demonstrates abnormal liver location, including “right extended” (B), “bilateral” (C), and “right” (D). (E) Graph depicting the scoring of pk1a morphants demonstrates a significant difference in the total number of abnormally localized livers (9% vs. 67%, p < 0.0001 by chi-square test), while the difference in livers limited to the right side is not significant (9% vs. 8%). Please see supplemental data for localization defects in other organs.

Expression Data

Expression Detail

Antibody Labeling

Phenotype Data

Fish:	WT + MO4-prickle1a
Knockdown Reagent:	MO4-prickle1a
Observed In:	liver
Stage:	Protruding-mouth

Phenotype Detail

Acknowledgments

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Reprinted from Developmental Biology, 351(2), Cui, S., Capecci, L.M., and Matthews, R.P., Disruption of planar cell polarity activity leads to developmental biliary defects, 229-241, Copyright (2011) with permission from Elsevier. Full text @ Dev. Biol.