Search Ontology:
ChEBI
EC 2.5.1.31 {ditrans,polycis-undecaprenyl-diphosphate synthase [(2E,6E)-farnesyl-diphosphate specific]} inhibitor
- Term ID
- CHEBI:85618
- Synonyms
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- (2E,6E)-farnesyl-diphosphate:isopentenyl-diphosphate cistransferase (adding 8 isopentenyl units) inhibitor
- (2E,6E)-farnesyl-diphosphate:isopentenyl-diphosphate cistransferase (adding 8 isopentenyl units) inhibitors
- bactoprenyl-diphosphate synthase inhibitor
- bactoprenyl-diphosphate synthase inhibitors
- ditrans,polycis-undecaprenyl-diphosphate synthase inhibitor
- ditrans,polycis-undecaprenyl-diphosphate synthase inhibitors
- ditrans,polycis-undecaprenylcistransferase inhibitor
- ditrans,polycis-undecaprenylcistransferase inhibitors
- EC 2.5.1.31 inhibitor
- EC 2.5.1.31 inhibitors
- EC 2.5.1.31 {ditrans,polycis-undecaprenyl-diphosphate synthase [(2E,6E)-farnesyl-diphosphate specific]} inhibitors
- undecaprenyl diphosphate synthetase inhibitor
- undecaprenyl diphosphate synthetase inhibitors
- undecaprenyl pyrophosphate synthase inhibitor
- undecaprenyl pyrophosphate synthase inhibitors
- undecaprenyl pyrophosphate synthetase inhibitor
- undecaprenyl pyrophosphate synthetase inhibitors
- undecaprenyl-diphosphate synthase inhibitor
- undecaprenyl-diphosphate synthase inhibitors
- UPP synthetase inhibitor
- UPP synthetase inhibitors
- Definition
- An EC 2.5.1.* (non-methyl-alkyl or aryl transferase) inhibitor that interferes with the action of ditrans,polycis-undecaprenyl-diphosphate synthase [(2E,6E)-farnesyl-diphosphate specific].
- References
- Ontology
- ChEBI ( EBI )
- is a type of
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- inverse has_role
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Phenotype
Phenotype resulting from EC 2.5.1.31 {ditrans,polycis-undecaprenyl-diphosphate synthase [(2E,6E)-farnesyl-diphosphate specific]} inhibitor
Phenotype where environments contain EC 2.5.1.31 {ditrans,polycis-undecaprenyl-diphosphate synthase [(2E,6E)-farnesyl-diphosphate specific]} inhibitor
Phenotype modified by environments containing EC 2.5.1.31 {ditrans,polycis-undecaprenyl-diphosphate synthase [(2E,6E)-farnesyl-diphosphate specific]} inhibitor
Human Disease Model