PUBLICATION

Transcriptional profiling of zebrafish identifies host factors controlling susceptibility to Shigella flexneri

Authors
Torraca, V., White, R.J., Sealy, I.M., Mazon-Moya, M., Duggan, G., Willis, A., Busch-Nentwich, E.M., Mostowy, S.
ID
ZDB-PUB-231222-15
Date
2023
Source
Disease models & mechanisms   17(1): (Journal)
Registered Authors
Busch-Nentwich, Elisabeth, Mostowy, Serge, Torraca, Vincenzo
Keywords
Shigella, Acod1, Gpr84, Host-pathogen, RNA-seq, Zebrafish
MeSH Terms
  • Animals
  • Dysentery, Bacillary*/genetics
  • Humans
  • Inflammation/microbiology
  • RNA, Messenger/genetics
  • RNA, Messenger/metabolism
  • Shigella flexneri/genetics
  • Shigella flexneri/metabolism
  • Zebrafish/genetics
  • Zebrafish/microbiology
PubMed
38131137 Full text @ Dis. Model. Mech.
Abstract
Shigella flexneri is a human-adapted pathovar of Escherichia coli that can invade the intestinal epithelium, causing inflammation and bacillary dysentery. Although an important human pathogen, the host response to S. flexneri has not been fully described. Zebrafish larvae represent a valuable model to study human infections in vivo. Here we use a Shigella-zebrafish infection model to generate mRNA expression profiles of host response to Shigella infection at the whole animal level. Immune response-related processes dominate the signature of early Shigella infection (6 hours post-infection). Consistent with its clearance from the host, the signature of late Shigella infection (24 hours post-infection) is significantly changed, and only a small set of immune-related genes remain differentially expressed, including acod1 and gpr84. Using mutant lines generated by ENU, CRISPR mutagenesis and F0 Crispants, we show that acod1- and gpr84-deficient larvae are more susceptible to Shigella infection. Together, these results highlight the power of zebrafish to model infection by bacterial pathogens and reveal the mRNA expression of the early (acutely infected) and late (clearing) host response to Shigella infection.
Genes / Markers
Figures
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Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping