PUBLICATION

Characterization of two novel ammonia transporters, Hiat1a and Hiat1b, in the teleost model system Danio rerio

Authors
Zhouyao, H., Zimmer, A.M., Fehsenfeld, S., Liebenstein, T., Richter, D.O., Begemann, G., Eck, P., Perry, S.F., Weihrauch, D.
ID
ZDB-PUB-220921-22
Date
2022
Source
The Journal of experimental biology   225(20): (Journal)
Registered Authors
Begemann, Gerrit, Perry, Steve F.
Keywords
Excretion, In situ hybridization, Morpholino, SIET, Zebrafish larvae
MeSH Terms
  • Ammonia/metabolism
  • Animals
  • Cation Transport Proteins*/metabolism
  • Larva/metabolism
  • Methylamines/metabolism
  • Morpholinos
  • Nitrogen/metabolism
  • Zebrafish*/genetics
  • Zebrafish*/metabolism
  • Zebrafish Proteins/genetics
  • Zebrafish Proteins/metabolism
PubMed
36124551 Full text @ J. Exp. Biol.
Abstract
Ammonia excretion in fish excretory epithelia is a complex interplay of multiple membrane transport proteins and mechanisms. Using the model system of zebrafish (Danio rerio) larvae, here we identified three paralogues of a novel ammonia transporter, hippocampus abundant transcript 1 (DrHIAT1), found also in most vertebrates. When functionally expressed in Xenopus laevis oocytes, DrHiat1a and DrHiat1b promoted methylamine uptake in a competitive manner with ammonia. In situ hybridization experiments showed that both transporters were expressed as early as the 4-cell stage in zebrafish embryos and could be identified in most tissues four days post fertilization. Larvae experiencing morpholino-mediated knockdown of DrHiat1b exhibited significantly lower whole body ammonia excretion rates compared to control larvae. Markedly decreased site-specific total ammonia excretion of up to 85% was observed in both the pharyngeal region (site of developing gills) and the yolk sac (region shown to have the highest NH4+ flux). This study is the first to identify especially DrHiat1b as an important contributor to ammonia excretion in larval zebrafish. Being evolutionarily conserved, these proteins are likely involved in multiple other general ammonia-handling mechanisms, making them worthy candidates for future studies on nitrogen regulation in fishes and across the animal kingdom.
Genes / Markers
Figures
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Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping