PUBLICATION

Reintroduction of DJ-1 in Müller Cells Inhibits Retinal Degeneration in the DJ-1 Deficient Retina

Authors
Gharbi, N., Røise, D., Førre, J.E., Edson, A.J., Hushagen, H.A., Tronci, V., Frøyset, A.K., Fladmark, K.E.
ID
ZDB-PUB-211230-3
Date
2021
Source
Antioxidants (Basel, Switzerland)   10(12): (Journal)
Registered Authors
Edson, Amanda, Fladmark, Kari E., Hushagen, Helena
Keywords
DJ-1, Müller cell, neurodegeneration, oxidative stress, retina
MeSH Terms
none
PubMed
34942966 Full text @ Antioxidants (Basel)
Abstract
The eye is continuously under oxidative stress due to high metabolic activity and reactive oxygen species generated by daily light exposure. The redox-sensitive protein DJ-1 has proven to be essential in order to protect retina and retinal pigment epithelium (RPE) from oxidative-stress-induced degeneration. Here, we analyzed the specific role of Müller cell DJ-1 in the adult zebrafish retina by re-establishing Müller-cell-specific DJ-1 expression in a DJ-1 knockout retina. Loss of DJ-1 resulted in an age-dependent retinal degeneration, including loss of cells in the ganglion cell layer, retinal thinning, photoreceptor disorganization and RPE cell dysfunction. The degenerative phenotype induced by the absence of DJ-1 was inhibited by solely expressing DJ-1 in Müller cells. The protective effect was dependent upon the cysteine-106 residue of DJ-1, which has been shown to be an oxidative sensor of DJ-1. In a label-free proteomics analysis of isolated retinas, we identified proteins differentially expressed after DJ-1 knockout, but with restored levels after Müller cell DJ-1 re-insertion. Our data show that Müller cell DJ-1 has a major role in protecting the retina from age-dependent oxidative stress.
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