PUBLICATION

CRISPR/Cas9-induced nos2b mutant zebrafish display behavioral abnormalities

Authors
Gao, L., Penglee, R., Huang, Y., Yi, X., Wang, X., Liu, L., Gong, X., Bao, B.
ID
ZDB-PUB-201120-152
Date
2020
Source
Genes, brain, and behavior   20(5): e12716 (Journal)
Registered Authors
Bao, Baolong
Keywords
none
MeSH Terms
  • Animals
  • Brain/metabolism
  • Gene Deletion
  • Homer Scaffolding Proteins/genetics
  • Homer Scaffolding Proteins/metabolism
  • Motor Activity*
  • Mutation
  • Nitric Oxide Synthase/genetics*
  • Nitric Oxide Synthase/metabolism
  • Synaptophysin/genetics
  • Synaptophysin/metabolism
  • Zebrafish
  • Zebrafish Proteins/genetics*
  • Zebrafish Proteins/metabolism
PubMed
33200539 Full text @ Genes Brain Behav.
Abstract
The immunomodulatory function of nitric oxide synthase (NOS2) has been extensively studied. However, some behavioral abnormalities caused by its mutations have been found in a few rodent studies, of which the molecular mechanism remains elusive. In this research, we generated nos2b gene knockout zebrafish (nos2b sou2/sou2 ) using CRISPR/Cas9 approach and investigated their behavioral and molecular changes by doing a series of behavioral detections, morphological measurements, and molecular analyses. We found that, compared with nos2b +/+ zebrafish, nos2b sou2/sou2 zebrafish exhibited enhanced motor activity; additionally, nos2b sou2/sou2 zebrafish were characterized by smaller brain size, abnormal structure of optic tectum, reduced mRNA level of presynaptic synaptophysin and postsynaptic homer1, and altered response to sodium nitroprusside/methylphenidate hydrochloride treatment. These findings will likely contribute to future studies of behavioral regulation.
Genes / Markers
Figures
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Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping