PUBLICATION

Mutations in Bcl9 and Pygo genes cause congenital heart defects by tissue-specific perturbation of Wnt/β-catenin signaling.

Authors
Cantù, C., Felker, A., Zimmerli, D., Prummel, K.D., Cabello, E.M., Chiavacci, E., Méndez-Acevedo, K.M., Kirchgeorg, L., Burger, S., Ripoll, J., Valenta, T., Hausmann, G., Vilain, N., Aguet, M., Burger, A., Panáková, D., Basler, K., Mosimann, C.
ID
ZDB-PUB-181028-1
Date
2018
Source
Genes & Development   32(21-22): 1443-1458 (Journal)
Registered Authors
Burger, Alexa, Burger, Sibylle, Chiavacci, Elena, Felker, Anastasia, Mosimann, Christian, Panáková, Daniela, Prummel, Karin
Keywords
CRISPR–Cas9, Wnt signaling, cardiovascular development, congenital heart disease, heart, transcription
MeSH Terms
  • Animals
  • Heart/embryology
  • Heart Defects, Congenital/genetics*
  • Intracellular Signaling Peptides and Proteins/genetics*
  • Mice
  • Mutation
  • Myocardium/metabolism
  • Transcription Factors/genetics*
  • Wnt Signaling Pathway*
  • Zebrafish/embryology
  • Zebrafish/genetics
  • Zebrafish Proteins/genetics*
  • beta Catenin/metabolism
PubMed
30366904 Full text @ Genes & Dev.
Abstract
Bcl9 and Pygopus (Pygo) are obligate Wnt/β-catenin cofactors in Drosophila, yet their contribution to Wnt signaling during vertebrate development remains unresolved. Combining zebrafish and mouse genetics, we document a conserved, β-catenin-associated function for BCL9 and Pygo proteins during vertebrate heart development. Disrupting the β-catenin-BCL9-Pygo complex results in a broadly maintained canonical Wnt response yet perturbs heart development and proper expression of key cardiac regulators. Our work highlights BCL9 and Pygo as selective β-catenin cofactors in a subset of canonical Wnt responses during vertebrate development. Moreover, our results implicate alterations in BCL9 and BCL9L in human congenital heart defects.
Genes / Markers
Figures
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Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping