PUBLICATION

Novel Genes Critical for Hypoxic Preconditioning in Zebrafish Are Regulators of Insulin and Glucose Metabolism

Authors
Manchenkov, T., Pasillas, M.P., Haddad, G.G., Imam, F.B.
ID
ZDB-PUB-150404-1
Date
2015
Source
G3 (Bethesda)   5(6): 1107-16 (Journal)
Registered Authors
Keywords
hormesis, hypoxia-ischemia, metabolic state, preconditioning, stress tolerance
Datasets
GEO:GSE68473
MeSH Terms
  • Animals
  • Embryo, Nonmammalian/metabolism
  • Gene Expression Profiling
  • Gene Knockdown Techniques
  • Genome
  • Glucose/metabolism*
  • Hypoxia/genetics*
  • Insulin/metabolism*
  • Phenotype
  • Real-Time Polymerase Chain Reaction
  • Reproducibility of Results
  • Stress, Physiological/genetics
  • Transcription, Genetic
  • Zebrafish/embryology
  • Zebrafish/genetics*
  • Zebrafish/metabolism*
  • Zebrafish Proteins/genetics
  • Zebrafish Proteins/metabolism
PubMed
25840431 Full text @ G3 (Bethesda)
Abstract
Severe hypoxia is a common cause of major brain, heart, and kidney injury in adults, children, and newborns. However, mild hypoxia can be protective against later, more severe hypoxia exposure via "hypoxic preconditioning", a phenomenon that is not yet fully understood. Accordingly, we have established and optimized an embryonic zebrafish model to study hypoxic preconditioning. Using a functional genomic approach, we used this zebrafish model to identify and validate five novel hypoxia-protective genes, including irs2, crtc3, and camk2g2, which have been previously implicated in metabolic regulation. These results extend our understanding of the mechanisms of hypoxic preconditioning and affirm the discovery potential of this novel vertebrate hypoxic stress model.
Genes / Markers
Figures
Show all Figures
Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping