PUBLICATION

Mutations in mitochondrial carrier family gene SLC25A38 cause nonsyndromic autosomal recessive congenital sideroblastic anemia

Authors
Guernsey, D.L., Jiang, H., Campagna, D.R., Evans, S.C., Ferguson, M., Kellogg, M.D., Lachance, M., Matsuoka, M., Nightingale, M., Rideout, A., Saint-Amant, L., Schmidt, P.J., Orr, A., Bottomley, S.S., Fleming, M.D., Ludman, M., Dyack, S., Fernandez, C.V., and Samuels, M.E.
ID
ZDB-PUB-110128-12
Date
2009
Source
Nature Genetics   41(6): 651-653 (Journal)
Registered Authors
Saint-Amant, Louis
Keywords
none
MeSH Terms
  • Anemia, Sideroblastic/genetics*
  • Animals
  • Carrier State
  • Family
  • Fishes/genetics
  • Heme/biosynthesis
  • Humans
  • Mitochondrial Membrane Transport Proteins/genetics*
  • Mutation*
  • Phenotype
  • Yeasts/genetics
PubMed
19412178 Full text @ Nat. Genet.
Abstract
The sideroblastic anemias are a heterogeneous group of congenital and acquired hematological disorders whose morphological hallmark is the presence of ringed sideroblasts--bone marrow erythroid precursors containing pathologic iron deposits within mitochondria. Here, by positional cloning, we define a previously unknown form of autosomal recessive nonsyndromic congenital sideroblastic anemia, associated with mutations in the gene encoding the erythroid specific mitochondrial carrier family protein SLC25A38, and demonstrate that SLC25A38 is important for the biosynthesis of heme in eukaryotes.
Genes / Markers
Figures
Show all Figures
Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping