PUBLICATION

CYP1A/Regucalcin Gene Expression and Edema Formation in Zebrafish Embryos Exposed to 2,3,7,8-Tetrachlorodibenzo-p-dioxin

Authors
Wu, Y.D., Jiang, L., Zhou, Z., Zheng, M.H., Zhang, J., and Liang, Y.
ID
ZDB-PUB-080515-8
Date
2008
Source
Bulletin of environmental contamination and toxicology   80(6): 482-486 (Journal)
Registered Authors
Keywords
2,3,7,8-Tetrachlorodibenzo-p-dioxin, Zebrafish, Edema, Gene expression
MeSH Terms
  • Animals
  • Calcium-Binding Proteins/genetics*
  • Calcium-Binding Proteins/metabolism
  • Cytochrome P-450 CYP1A1/genetics*
  • Cytochrome P-450 CYP1A1/metabolism
  • Dose-Response Relationship, Drug
  • Edema/chemically induced*
  • Edema/embryology
  • Edema/physiopathology
  • Embryo, Nonmammalian/drug effects*
  • Embryo, Nonmammalian/pathology
  • Embryo, Nonmammalian/physiopathology
  • Gene Expression Regulation, Developmental/drug effects*
  • Heart Rate/drug effects
  • Longevity/drug effects
  • Pericardial Effusion/chemically induced
  • Pericardial Effusion/embryology
  • Pericardial Effusion/pathology
  • RNA, Messenger/metabolism
  • Teratogens/toxicity*
  • Zebrafish
  • Zebrafish Proteins/genetics*
  • Zebrafish Proteins/metabolism
PubMed
18470470 Full text @ Bull. Environ. Contam. Toxicol.
CTD
18470470
Abstract
In this study, zebrafish eggs were exposed to a relatively low concentration (50 pg/mL) of 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) for 72 h and then transferred to vehicle/TCDD-free water for the remainder of the experiments. Mortality, heart rates, edema severity, CYP1A, and regucalcin gene expressions were investigated to study TCDD-induced toxicity in zebrafish during the early life stage. Results indicated that the 50 pg/mL TCDD caused severe and visible developmental toxicity. Further research of the long term and low concentration of TCDD exposure is required.
Genes / Markers
Figures
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Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping