PUBLICATION

L1.1 is involved in spinal cord regeneration in adult zebrafish

Authors
Becker, C.G., Lieberoth, B.C., Morellini, F., Feldner, J., Becker, T., and Schachner, M.
ID
ZDB-PUB-040913-2
Date
2004
Source
The Journal of neuroscience : the official journal of the Society for Neuroscience   24(36): 7837-7842 (Journal)
Registered Authors
Becker, Catherina G., Becker, Thomas, Feldner, Julia, Lieberoth, Bettina, Schachner, Melitta
Keywords
regeneration; cell recognition molecule; spinal cord; brainstem; teleost; Danio rerio
MeSH Terms
  • Animals
  • Axons/metabolism
  • Brain Stem/physiopathology
  • Cordotomy
  • Drug Implants
  • Gelatin Sponge, Absorbable
  • Morpholines/pharmacology
  • Nerve Regeneration/drug effects
  • Nerve Regeneration/physiology*
  • Oligodeoxyribonucleotides/pharmacology
  • Recovery of Function
  • Single-Blind Method
  • Spinal Cord/physiology*
  • Spinal Cord Injuries/physiopathology*
  • Swimming
  • Zebrafish/physiology*
PubMed
15356195 Full text @ J. Neurosci.
Abstract
Adult zebrafish, in contrast to mammals, regrow axons descending from the brainstem after spinal cord transection. L1.1, a homolog of the mammalian recognition molecule L1, is upregulated by brainstem neurons during axon regrowth. However, its functional relevance for regeneration is unclear. Here, we show with a novel morpholino-based approach that reducing L1.1 protein expression leads to impaired locomotor recovery as well as reduced regrowth and synapse formation of axons of supraspinal origin after spinal cord transection. This indicates that L1.1 contributes to successful regrowth of axons from the brainstem and locomotor recovery after spinal cord transection in adult zebrafish.
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