Person

Traver, David

Person ID
ZDB-PERS-010126-6
Email
dtraver@ucsd.edu
URL
http://www.biology.ucsd.edu/labs/traver/index.html
Affiliation
Traver Lab
Address
Assistant Professor Section of Cell and Developmental Biology University of California, San Diego 9500 Gilman Drive Natural Sciences Building 6107 La Jolla, CA 92093-0380 USA
Country
United States
Phone
(858) 822-4593
Fax
(858) 534-5457
ORCID ID
Biography and Research Interest
Publications
Non-Zebrafish Publications
BitMansour, A., S. M. Burns, D. Traver, K. Akashi, C. H. Contag, I. L. Weissman, and J. M. Y. Brown. 2002. Myeloid progenitors protect against invasive aspergillosis and Pseudomonas aeruginosa infection following hematopoietic stem cell transplantation. Blood, In press.

NaNakorn, T.*, D. Traver*, K. Akashi, and I. L. Weissman. 2002. Myeloerythroid-restricted progenitors are sufficient to confer radioprotection and provide the majority of day 8 CFU-S. J. Clin. Invest., 109(12): 1579-1585.

Traver, D., T. Miyamoto, J. Christensen, K. Akashi, and I. L. Weissman. 2001. Fetal liver myelopoiesis occurs through distinct, prospectively isolatable progenitor subsets. Blood, 98(3): 627-635.

DiMartino, J.*, L. Selleri*, D. Traver*, M. Firpo, R. Warnke, J. Rhee, S. O'Garman, I. L. Weissman, and M. L. Cleary. 2001. The Hox cofactor and proto-oncogene Pbx1 is required for maintenance of definitive hematopoiesis in the fetal liver. Blood, 98(3): 618-626.

Manz, M. G., D. Traver, T. Miyamoto, I. L. Weissman, and K. Akashi. 2001. Dendritic cell potentials of early lymphoid and myeloid progenitors. Blood, 97: 3333-3341.

Traver, D.*, K. Akashi*, M. Manz*, M. Merad, T. Miyamoto, E. G. Engleman, and I. L. Weissman. 2000. Development of CD8a-Positive dendritic cells from a common myeloid progenitor. Science, 290: 2152-2154.

Akashi, K.*, D. Traver*, T. Miyamoto, and I.L. Weissman. 2000. A clonogenic common myeloid progenitor that gives rise to all myeloid lineages. Nature 404: 193-197.

Traver, D., K. Akashi, I.L. Weissman, and E. Lagasse. 1998. Mice defective in two apoptosis pathways in the myeloid lineage develop acute myeloblastic leukemia. Immunity 9, no. 1:47-57.