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Figure 7
Anorexigenic signals in hypothalamic neural circuits promoted by α-MSH expression in zebrafish. (A) A proposed mechanism that α-MSH mediating food intake and energy expenditure via anorexigenic molecules manipulation which can reversed by α-MSH depletion. (B) Schematic presentation of anorexigenic factors (bdnf, sim1a, crhb, and trh) enhancement within the paraventricular nucleus (PVN) and the ventromedial hypothalamus (VMN) by α-MSH resulting in a dramatic outcome with the anorexigenic phenomenon.
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