ZFIN Image: Yabe et al., 2012, Fig. 1

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Figure Caption

Fig. 1 msgn1 and spt are required for the differentiation of tail PSM in zebrafish. (A)-(G) Morphologies of wild-type zebrafish embryos injected with 5mis MO (A) or msgn1 MO (B), spt heterozygous embryo injected with msgn1 MO (C), and spt homozygous embryo injected with 5mis MO (D) or msgn1 MO (E) at 24 hpf. Ventral views of the tailbud region of msgn1 MO-injected wild-type (F) and spt heterozygous (G) embryos are also shown. Broken lines indicate the tail somites. (H)-(L) Defective tail myogenesis in msgn1;spt double-deficient embryos. Expression of myod, a myogenic gene, in the tail region of the msgn1-injected spt mutant was severely reduced (L) compared with that in the wild-type or single-deficient embryo (H)-(K). Embryos were fixed at 24 hpf for in situ hybridization.

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Acknowledgments

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Reprinted from Developmental Biology, 370(2), Yabe, T., and Takada, S., Mesogenin causes embryonic mesoderm progenitors to differentiate during development of zebrafish tail somites, 213-222, Copyright (2012) with permission from Elsevier. Full text @ Dev. Biol.